Hypertension: MechanismsJohn H. Laragh Yorke Medical Books, 1975 - Medical |
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Page 178
... norepinephrine from L - dopa . Chronic administration of reserpine did not change the endogenous excretion of norepinephrine , dopamine or VMA ; however , the decline of norepinephrine specific activity in the urine of reserpine ...
... norepinephrine from L - dopa . Chronic administration of reserpine did not change the endogenous excretion of norepinephrine , dopamine or VMA ; however , the decline of norepinephrine specific activity in the urine of reserpine ...
Page 181
... norepinephrine were actually decreased [ 4 ] . We are unable to find differences in norepinephrine or VMA excretion in these animals . * Thus , whether or not catecholamine excretion was increased , decreased or normal , one could not ...
... norepinephrine were actually decreased [ 4 ] . We are unable to find differences in norepinephrine or VMA excretion in these animals . * Thus , whether or not catecholamine excretion was increased , decreased or normal , one could not ...
Page 191
... norepinephrine in hypertension may indicate that these patients have enhanced release of newly synthesized norepinephrine . In hypertensive patients , recent findings of increased plasma norepinephrine cast doubt that norepi- nephrine ...
... norepinephrine in hypertension may indicate that these patients have enhanced release of newly synthesized norepinephrine . In hypertensive patients , recent findings of increased plasma norepinephrine cast doubt that norepi- nephrine ...
Contents
A Viewpoint Concerning the Enigma of Hypertension | 25 |
The Control of Renin Release X | 53 |
The Control of Aldosterone Secretion in Normal | 87 |
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13 other sections not shown
Common terms and phrases
abnormal adrenal aldosterone secretion Amer J Physiol angiotensin angiotensin II antihypertensive arterial pressure arterial renin arteriolar arterioles blood pressure blood volume cardiac output catecholamines cent changes chronic renal failure Circ Res Circulation Clin Invest clinical constriction contralateral decreased diabetic diastolic diuretic dogs effect essential hypertension experimental factors Figure hemodynamic high renin hormonal hyper hypertensive patients increased infusion intrarenal juxtaglomerular cells kidney LARAGH LARAGH JH left ventricular lesions low renin macula densa malignant hypertension mechanism metabolism myocardial nephrectomy norepinephrine normal subjects normotensive observed papilla patients with essential peripheral resistance plasma aldosterone plasma renin activity plasma renin concentration primary aldosteronism propranolol reduced relationship renal artery stenosis renal blood flow renal disease renal function renal hypertension renal plasma flow renal vein renin renin essential hypertension renin levels renin release renin secretion renin-angiotensin system renovascular hypertension response sodium balance sodium depletion sodium excretion suppression sympathetic nerve syndrome tension toxemia urinary venous
References to this book
Principles of Anatomy and Physiology Gerard J. Tortora,Nicholas Peter Anagnostakos Snippet view - 1981 |