Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
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Page 231
... Amyloid is a pathologic proteinaceous sub- stance , deposited between cells in various tissues and organs of the body in a wide variety of clinical settings . Because amyloid deposition appears so in- sidiously and sometimes ...
... Amyloid is a pathologic proteinaceous sub- stance , deposited between cells in various tissues and organs of the body in a wide variety of clinical settings . Because amyloid deposition appears so in- sidiously and sometimes ...
Page 232
... amyloid - associated ) protein that is synthesized in the liver and circulates in association with the HDL3 subclass of lipoproteins . Several other biochemically distinct proteins have been found in amyloid deposits in a variety of ...
... amyloid - associated ) protein that is synthesized in the liver and circulates in association with the HDL3 subclass of lipoproteins . Several other biochemically distinct proteins have been found in amyloid deposits in a variety of ...
Page 236
... amyloid deposits . Under polarized light , the Congo red - stained amyloid shows a green bire- fringence ( Fig . 6-43 ) . This reaction is shared by all forms of amyloid and is due to the cross - beta- pleated configuration of amyloid ...
... amyloid deposits . Under polarized light , the Congo red - stained amyloid shows a green bire- fringence ( Fig . 6-43 ) . This reaction is shared by all forms of amyloid and is due to the cross - beta- pleated configuration of amyloid ...
Contents
Cellular Injury and Cellular Death | 4 |
CHAPTER | 20 |
Normal | 35 |
Copyright | |
17 other sections not shown
Common terms and phrases
abnormal acid activation acute adhesion amyloidosis anemia antibodies antigens aortic apoptosis arteries associated atherosclerosis autoimmune bacterial binding blood bone marrow cancer carcinoma cardiac cause CD4+ cellular changes Chapter chromosome chronic clinical collagen congenital coronary cytokines damage death defects deficiency develop dilatation disorders edema effects endothelial cells endothelium enzymes epithelial fibrosis Figure function gene genetic growth factor heart disease hemolytic hemorrhage histologic host human hypertension immune increased induced infection inflammation inflammatory injury intracellular involved ischemic lesions leukocytes lipid liver lung lymph nodes lymphocytes lymphomas lysosomal macrophages major malignant mechanisms mediators membrane metabolism molecular molecules monocytes MORPHOLOGY muscle mutations myocardial infarction myocardium necrosis neoplasms neutrophils normal occur oncogenes organs pathogenesis patients phagocytosis plaque plasma platelet produce proliferation protein pulmonary reaction receptor red cells renal result risk serum skin syndrome synthesis T-cell thrombi thrombosis tion tissue tumor cells valve vascular vasculitis ventricular vessels viral virus vitamin