Robbins' Pathologic Basis of Disease, Volume 1Presenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 447
In the last analysis, the best way to make the diagnosis of osteomalacia in most
instances is a therapeutic trial of vitamin D and calcium. The serum calcium and
25-OH-D levels are not usually helpful because they may be normal or low.
In the last analysis, the best way to make the diagnosis of osteomalacia in most
instances is a therapeutic trial of vitamin D and calcium. The serum calcium and
25-OH-D levels are not usually helpful because they may be normal or low.
Page 448
The changes have taken the form mostly of cell atrophy with increased deposits
of electron-dense inclusions, most likely lipofuscin and secondary lysosomes.
Retinal pigmentary degeneration similar to that seen in severe vitamin A
deficiency ...
The changes have taken the form mostly of cell atrophy with increased deposits
of electron-dense inclusions, most likely lipofuscin and secondary lysosomes.
Retinal pigmentary degeneration similar to that seen in severe vitamin A
deficiency ...
Page 449
The biochemical events in the carboxylation of vitamin K- dependent proteins.
intestinal flora of the breast-fed infant produces less of the vitamin than the flora
of the formula-fed infant. Moreover, the reserves of vitamin K in the postnatal
period ...
The biochemical events in the carboxylation of vitamin K- dependent proteins.
intestinal flora of the breast-fed infant produces less of the vitamin than the flora
of the formula-fed infant. Moreover, the reserves of vitamin K in the postnatal
period ...
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Contents
Cellular Injury and Adaptation l | 9 |
Inflammation and Repair | 39 |
Fluid and Hemodynamic Derangements | 87 |
Copyright | |
28 other sections not shown
Common terms and phrases
abnormal acid activity acute adult agents amyloid amyloidosis antibodies antigens arteries atherosclerosis bacterial blood bone cancer carcinogens carcinoma cardiac cause cellular changes chemical chromosome chronic clinical collagen common complex damage death defects deficiency develop diagnosis disease disorders drugs edema effects endothelial cells endothelium Engl enzymes epithelial fatty fever fibroblasts fibrosis Figure fluid focal function gene genetic granulomas growth factor hemorrhage hepatic histologic host human immune immunologic increased induce infants infarction infection infiltration inflammation inflammatory injury interstitial intestinal intracellular involved lesions leukemia leukocytes levels lipid liver lung lymph nodes lymphocytes lysosomal macrophages malignant mechanisms mediators membrane metabolism monocytes mucosa muscle mutations necrosis necrotic neoplasms neutrophils normal occur oncogenes organs parasites pathogenesis patients phagocytic phagocytosis plasma platelet produce proliferation protein pulmonary reaction receptor renal respiratory response result risk serum skin specific syndrome synthesis tion tissue toxic tumor tumor cells ulcer usually vascular vessels viral virus viruses vitamin
Bibliographic information
| Title | Robbins' Pathologic Basis of Disease, Volume 1 Robbins' pathologic basis of disease, Stanley Leonard Robbins |
| Contributors | Stanley Leonard Robbins, Ramzi S. Cotran, Vinay Kumar |
| Edition | 4 |
| Publisher | Saunders, 1989 |
| Original from | University of Minnesota |
| Digitized | 21 Jan 2010 |
| ISBN | 0721623026, 9780721623023 |
| Length | 1519 pages |
| Export Citation | BiBTeX EndNote RefMan |