Robbins' Pathologic Basis of Disease, Volume 1Presenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
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Page 168
... monocytes Supports growth of pluripotential hematopoietic stem cells ; growth factor for mast cells Growth and differentiation factor for activated B and T cells ; growth factor for mast cells ; increases expression of HLA class II ...
... monocytes Supports growth of pluripotential hematopoietic stem cells ; growth factor for mast cells Growth and differentiation factor for activated B and T cells ; growth factor for mast cells ; increases expression of HLA class II ...
Page 230
... monocyte infection may be direct transfer of HIV from the monocytes to CD4 + T cells when the two cells come in close contact in the pro- cess of antigen presentation . Low - level chronic or latent infection of T cells and monocytes is ...
... monocyte infection may be direct transfer of HIV from the monocytes to CD4 + T cells when the two cells come in close contact in the pro- cess of antigen presentation . Low - level chronic or latent infection of T cells and monocytes is ...
Page 566
... monocytes to adhere to endothelium or subendothelial connective tissue . Factors released from activated platelets or monocytes then cause migration of smooth muscle cells from media into the intima , followed by prolifer- ation ...
... monocytes to adhere to endothelium or subendothelial connective tissue . Factors released from activated platelets or monocytes then cause migration of smooth muscle cells from media into the intima , followed by prolifer- ation ...
Contents
Cellular Injury and Adaptation | 1 |
Inflammation and Repair | 39 |
Fluid and Hemodynamic Derangements | 87 |
Copyright | |
28 other sections not shown
Common terms and phrases
abnormal acid activity acute adult agents amyloid amyloidosis antibodies antigens arteries atherosclerosis bacterial blood bone cancer carcinogens carcinoma cardiac cause cellular changes chemical chromosome chronic clinical collagen complex damage death defects deficiency develop diagnosis disease disorders drugs edema effects endothelium Engl enzymes epithelial fatty fever fibroblasts fibrosis Figure fluid function gene genetic granulomas growth factor hemorrhage hepatic histologic host human immune immunologic increased induce infants infarction infection infiltration inflammation inflammatory injury interstitial intestinal intracellular involved lesions leukemia leukocytes levels lipid liver lung lymph nodes lymphocytes lysosomal macrophages malignant mechanisms mediators membrane metabolism monocytes mucosa muscle mutations necrosis necrotic neoplasms neutrophils normal occur oncogenes organs parasites pathogenesis patients phagocytic phagocytosis plasma platelet produce proliferation protein pulmonary reaction receptor renal respiratory response result risk serum skin specific syndrome synthesis tion tissue toxic tumor tumor cells ulcer usually vascular vessels viral virus viruses vitamin vitamin D