Robbins' Pathologic Basis of Disease, Volume 1Presenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
Results 1-3 of 85
Page 3
... injury defies compi- lation . Simple chemicals such as glucose or salt in hy- pertonic concentrations may cause cell injury by de- ranging the fluid and electrolyte homeostasis of cells . Even oxygen , in high concentrations , is ...
... injury defies compi- lation . Simple chemicals such as glucose or salt in hy- pertonic concentrations may cause cell injury by de- ranging the fluid and electrolyte homeostasis of cells . Even oxygen , in high concentrations , is ...
Page 4
... injury ( Fig . 1–2 ) . Lack of oxygen clearly under- lies the pathogenesis of cell injury in ischemia , but it also is clear that partially reduced activated oxygen species are important mediators of cell death in many pathologic ...
... injury ( Fig . 1–2 ) . Lack of oxygen clearly under- lies the pathogenesis of cell injury in ischemia , but it also is clear that partially reduced activated oxygen species are important mediators of cell death in many pathologic ...
Page 501
... injury encountered in clinical prac- tice . Much detail on mechanical injuries is unneces- sary in general pathology , since only rarely do such cases necessitate pathologic study save for legal pur- poses . Changes in atmospheric ...
... injury encountered in clinical prac- tice . Much detail on mechanical injuries is unneces- sary in general pathology , since only rarely do such cases necessitate pathologic study save for legal pur- poses . Changes in atmospheric ...
Contents
Cellular Injury and Adaptation | 1 |
Inflammation and Repair | 39 |
Fluid and Hemodynamic Derangements | 87 |
Copyright | |
28 other sections not shown
Common terms and phrases
abnormal acid activity acute adult agents amyloid amyloidosis antibodies antigens arteries atherosclerosis bacterial blood bone cancer carcinogens carcinoma cardiac cause cellular changes chemical chromosome chronic clinical collagen complex damage death defects deficiency develop diagnosis disease disorders drugs edema effects endothelium Engl enzymes epithelial fatty fever fibroblasts fibrosis Figure fluid function gene genetic granulomas growth factor hemorrhage hepatic histologic host human immune immunologic increased induce infants infarction infection infiltration inflammation inflammatory injury interstitial intestinal intracellular involved lesions leukemia leukocytes levels lipid liver lung lymph nodes lymphocytes lysosomal macrophages malignant mechanisms mediators membrane metabolism monocytes mucosa muscle mutations necrosis necrotic neoplasms neutrophils normal occur oncogenes organs parasites pathogenesis patients phagocytic phagocytosis plasma platelet produce proliferation protein pulmonary reaction receptor renal respiratory response result risk serum skin specific syndrome synthesis tion tissue toxic tumor tumor cells ulcer usually vascular vessels viral virus viruses vitamin vitamin D