Robbins' Pathologic Basis of Disease, Volume 1Presenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
Results 1-3 of 73
Page 182
... initial peri- vascular mononuclear cell infiltrate is replaced by granulomas over a period of two to three weeks . Delayed hypersensitivity reactions are initiated by specifically sensitized T lymphocytes , generated dur- ing the initial ...
... initial peri- vascular mononuclear cell infiltrate is replaced by granulomas over a period of two to three weeks . Delayed hypersensitivity reactions are initiated by specifically sensitized T lymphocytes , generated dur- ing the initial ...
Page 185
... initial points of contact of the host im- mune recognition and effector mechanisms . The re- sulting reaction is termed " hyperacute rejection . " The antidonor antibodies responsible for hyperacute rejection may be directed against HLA ...
... initial points of contact of the host im- mune recognition and effector mechanisms . The re- sulting reaction is termed " hyperacute rejection . " The antidonor antibodies responsible for hyperacute rejection may be directed against HLA ...
Page 633
... initial acute attack it is the myocarditis that is most threaten- ing , causing arrhythmias ( particularly atrial fibrilla- tion ) and prolongation of the electrocardiographic P - R interval . With fibrillation , auricular thrombi are ...
... initial acute attack it is the myocarditis that is most threaten- ing , causing arrhythmias ( particularly atrial fibrilla- tion ) and prolongation of the electrocardiographic P - R interval . With fibrillation , auricular thrombi are ...
Contents
Cellular Injury and Adaptation | 1 |
Inflammation and Repair | 39 |
Fluid and Hemodynamic Derangements | 87 |
Copyright | |
28 other sections not shown
Common terms and phrases
abnormal acid activity acute adult agents amyloid amyloidosis antibodies antigens arteries atherosclerosis bacterial blood bone cancer carcinogens carcinoma cardiac cause cellular changes chemical chromosome chronic clinical collagen complex damage death defects deficiency develop diagnosis disease disorders drugs edema effects endothelium Engl enzymes epithelial fatty fever fibroblasts fibrosis Figure fluid function gene genetic granulomas growth factor hemorrhage hepatic histologic host human immune immunologic increased induce infants infarction infection infiltration inflammation inflammatory injury interstitial intestinal intracellular involved lesions leukemia leukocytes levels lipid liver lung lymph nodes lymphocytes lysosomal macrophages malignant mechanisms mediators membrane metabolism monocytes mucosa muscle mutations necrosis necrotic neoplasms neutrophils normal occur oncogenes organs parasites pathogenesis patients phagocytic phagocytosis plasma platelet produce proliferation protein pulmonary reaction receptor renal respiratory response result risk serum skin specific syndrome synthesis tion tissue toxic tumor tumor cells ulcer usually vascular vessels viral virus viruses vitamin vitamin D