Robbins' Pathologic Basis of Disease, Volume 1Presenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
Results 1-3 of 87
Page 12
... induce mutations in the genetic code , which , if not repaired , induce cellular derangements as well as inhibition of DNA replication . All three oxygen - de- rived radicals are involved in such DNA damage.36 Lipid peroxidation is one ...
... induce mutations in the genetic code , which , if not repaired , induce cellular derangements as well as inhibition of DNA replication . All three oxygen - de- rived radicals are involved in such DNA damage.36 Lipid peroxidation is one ...
Page 273
... induce clonal proliferation of initiated cells and alter their differentiation programs by activating enzymes that are part of the physiologic signal transduction pathways . The two - stage initiation - promotion sequence is fol- lowed ...
... induce clonal proliferation of initiated cells and alter their differentiation programs by activating enzymes that are part of the physiologic signal transduction pathways . The two - stage initiation - promotion sequence is fol- lowed ...
Page 646
... induce this form of CMP , including cardiac amyloidosis , sar- coidosis ( discussed elsewhere , p . 427 ) , endocardial fibroelastosis , endomyocardial fibrosis , and Loeffler's endocarditis . Cardiac amyloidosis ( p . 219 ) , you ...
... induce this form of CMP , including cardiac amyloidosis , sar- coidosis ( discussed elsewhere , p . 427 ) , endocardial fibroelastosis , endomyocardial fibrosis , and Loeffler's endocarditis . Cardiac amyloidosis ( p . 219 ) , you ...
Contents
Cellular Injury and Adaptation | 1 |
Inflammation and Repair | 39 |
Fluid and Hemodynamic Derangements | 87 |
Copyright | |
28 other sections not shown
Common terms and phrases
abnormal acid activity acute adult agents amyloid amyloidosis antibodies antigens arteries atherosclerosis bacterial blood bone cancer carcinogens carcinoma cardiac cause cellular changes chemical chromosome chronic clinical collagen complex damage death defects deficiency develop diagnosis disease disorders drugs edema effects endothelium Engl enzymes epithelial fatty fever fibroblasts fibrosis Figure fluid function gene genetic granulomas growth factor hemorrhage hepatic histologic host human immune immunologic increased induce infants infarction infection infiltration inflammation inflammatory injury interstitial intestinal intracellular involved lesions leukemia leukocytes levels lipid liver lung lymph nodes lymphocytes lysosomal macrophages malignant mechanisms mediators membrane metabolism monocytes mucosa muscle mutations necrosis necrotic neoplasms neutrophils normal occur oncogenes organs parasites pathogenesis patients phagocytic phagocytosis plasma platelet produce proliferation protein pulmonary reaction receptor renal respiratory response result risk serum skin specific syndrome synthesis tion tissue toxic tumor tumor cells ulcer usually vascular vessels viral virus viruses vitamin vitamin D