Robbins' Pathologic Basis of Disease, Volume 1Presenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
Results 1-3 of 74
Page 32
... increased RNA synthesis , increased protein synthesis , and the for- mation of a greater number of all organelles , including mitochondria , sarcoplasmic reticulum , and especially myofibrils . Whatever the exact mechanism of ...
... increased RNA synthesis , increased protein synthesis , and the for- mation of a greater number of all organelles , including mitochondria , sarcoplasmic reticulum , and especially myofibrils . Whatever the exact mechanism of ...
Page 88
... INCREASED HYDROSTATIC PRESSURE . An in- crease in hydrostatic pressure in the venular end of the microcirculation raises the filtration pressure and at the same time opposes the oncotic pressure of the plasma proteins , and so flow from ...
... INCREASED HYDROSTATIC PRESSURE . An in- crease in hydrostatic pressure in the venular end of the microcirculation raises the filtration pressure and at the same time opposes the oncotic pressure of the plasma proteins , and so flow from ...
Page 850
... increased capacity to secrete acid and pepsin , ( 2 ) increased responsiveness to stimuli of acid secretion , and ( 3 ) more rapid gastric emptying.50 However , not all these characteristics are present in every patient . In general ...
... increased capacity to secrete acid and pepsin , ( 2 ) increased responsiveness to stimuli of acid secretion , and ( 3 ) more rapid gastric emptying.50 However , not all these characteristics are present in every patient . In general ...
Contents
Cellular Injury and Adaptation | 1 |
Inflammation and Repair | 39 |
Fluid and Hemodynamic Derangements | 87 |
Copyright | |
28 other sections not shown
Common terms and phrases
abnormal acid activity acute adult agents amyloid amyloidosis antibodies antigens arteries atherosclerosis bacterial blood bone cancer carcinogens carcinoma cardiac cause cellular changes chemical chromosome chronic clinical collagen complex damage death defects deficiency develop diagnosis disease disorders drugs edema effects endothelium Engl enzymes epithelial fatty fever fibroblasts fibrosis Figure fluid function gene genetic granulomas growth factor hemorrhage hepatic histologic host human immune immunologic increased induce infants infarction infection infiltration inflammation inflammatory injury interstitial intestinal intracellular involved lesions leukemia leukocytes levels lipid liver lung lymph nodes lymphocytes lysosomal macrophages malignant mechanisms mediators membrane metabolism monocytes mucosa muscle mutations necrosis necrotic neoplasms neutrophils normal occur oncogenes organs parasites pathogenesis patients phagocytic phagocytosis plasma platelet produce proliferation protein pulmonary reaction receptor renal respiratory response result risk serum skin specific syndrome synthesis tion tissue toxic tumor tumor cells ulcer usually vascular vessels viral virus viruses vitamin vitamin D