Robbins' Pathologic Basis of Disease, Volume 1Presenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 311
nism to establish itself in a host, but its ability to produce an infectious disease
relates to its virulence. The terms resistance and virulence are broad
generalizations based on the complex host-parasite interplay that takes place at
the molecular ...
nism to establish itself in a host, but its ability to produce an infectious disease
relates to its virulence. The terms resistance and virulence are broad
generalizations based on the complex host-parasite interplay that takes place at
the molecular ...
Page 397
Some cycles are based on alternating sexual and asexual modes of reproduction
(e.g., Toxoplasma gametes differentiate only in the gut of feline hosts that have
fed on other host species bearing asexual parasite forms). One marvels at the ...
Some cycles are based on alternating sexual and asexual modes of reproduction
(e.g., Toxoplasma gametes differentiate only in the gut of feline hosts that have
fed on other host species bearing asexual parasite forms). One marvels at the ...
Page 412
and malabsorptive changes in the gut (Strongyloides stercoralis); or (5) inducing
host hypersensitivity reactions (many helminths). Hypersensitivity reactions are
often caused by the migratory larval stages of those helminths that must invade ...
and malabsorptive changes in the gut (Strongyloides stercoralis); or (5) inducing
host hypersensitivity reactions (many helminths). Hypersensitivity reactions are
often caused by the migratory larval stages of those helminths that must invade ...
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Contents
Cellular Injury and Adaptation l | 9 |
Inflammation and Repair | 39 |
Fluid and Hemodynamic Derangements | 87 |
Copyright | |
28 other sections not shown
Common terms and phrases
abnormal acid activity acute adult agents amyloid amyloidosis antibodies antigens arteries atherosclerosis bacterial blood bone cancer carcinogens carcinoma cardiac cause cellular changes chemical chromosome chronic clinical collagen common complex damage death defects deficiency develop diagnosis disease disorders drugs edema effects endothelial cells endothelium Engl enzymes epithelial fatty fever fibroblasts fibrosis Figure fluid focal function gene genetic granulomas growth factor hemorrhage hepatic histologic host human immune immunologic increased induce infants infarction infection infiltration inflammation inflammatory injury interstitial intestinal intracellular involved lesions leukemia leukocytes levels lipid liver lung lymph nodes lymphocytes lysosomal macrophages malignant mechanisms mediators membrane metabolism monocytes mucosa muscle mutations necrosis necrotic neoplasms neutrophils normal occur oncogenes organs parasites pathogenesis patients phagocytic phagocytosis plasma platelet produce proliferation protein pulmonary reaction receptor renal respiratory response result risk serum skin specific syndrome synthesis tion tissue toxic tumor tumor cells ulcer usually vascular vessels viral virus viruses vitamin