Robbins' Pathologic Basis of Disease, Volume 1Presenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Results 1-3 of 87
Page 80
... collagen ( see text ) . ( Adapted from Krieg , T. , et al .: Molecular defects of collagen metabolism in the Ehlers - Danlos syndrome . Int . J. Dermatol . 20 : 415 , 1981. ) INJURY VASCULAR AND CELLULAR RESPONSE ACUTE INFLAMMATORY ...
... collagen ( see text ) . ( Adapted from Krieg , T. , et al .: Molecular defects of collagen metabolism in the Ehlers - Danlos syndrome . Int . J. Dermatol . 20 : 415 , 1981. ) INJURY VASCULAR AND CELLULAR RESPONSE ACUTE INFLAMMATORY ...
Page 156
... collagen . Because abnor- malities of collagen are fundamental in the pathogen- esis of EDS it would be advisable to review collagen structure and synthesis , discussed in Chapter 2. We should recall that there are at least 10 ...
... collagen . Because abnor- malities of collagen are fundamental in the pathogen- esis of EDS it would be advisable to review collagen structure and synthesis , discussed in Chapter 2. We should recall that there are at least 10 ...
Page 259
... collagen is the most abundant protein of the interstitial extracellular matrix . However , the type of collagen varies ; type I collagen is the predominant type ( and is usually co- distributed with type III collagen ) in all ...
... collagen is the most abundant protein of the interstitial extracellular matrix . However , the type of collagen varies ; type I collagen is the predominant type ( and is usually co- distributed with type III collagen ) in all ...
Contents
Cellular Injury and Adaptation | 1 |
Inflammation and Repair | 39 |
Fluid and Hemodynamic Derangements | 87 |
Copyright | |
28 other sections not shown
Common terms and phrases
abnormal acid activity acute adult agents amyloid amyloidosis antibodies antigens arteries atherosclerosis bacterial blood bone cancer carcinogens carcinoma cardiac cause cellular changes chemical chromosome chronic clinical collagen complex damage death defects deficiency develop diagnosis disease disorders drugs edema effects endothelium Engl enzymes epithelial fatty fever fibroblasts fibrosis Figure fluid function gene genetic granulomas growth factor hemorrhage hepatic histologic host human immune immunologic increased induce infants infarction infection infiltration inflammation inflammatory injury interstitial intestinal intracellular involved lesions leukemia leukocytes levels lipid liver lung lymph nodes lymphocytes lysosomal macrophages malignant mechanisms mediators membrane metabolism monocytes mucosa muscle mutations necrosis necrotic neoplasms neutrophils normal occur oncogenes organs parasites pathogenesis patients phagocytic phagocytosis plasma platelet produce proliferation protein pulmonary reaction receptor renal respiratory response result risk serum skin specific syndrome synthesis tion tissue toxic tumor tumor cells ulcer usually vascular vessels viral virus viruses vitamin vitamin D