Robbins' Pathologic Basis of Disease, Volume 1Presenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 128
... chromosome 2 and a translocated chromosome 5 may be formed . Such a gamete would be unbalanced since it would not contain the normal complement of genetic mate- rial . Subsequent fertilization by a normal gamete would lead to the ...
... chromosome 2 and a translocated chromosome 5 may be formed . Such a gamete would be unbalanced since it would not contain the normal complement of genetic mate- rial . Subsequent fertilization by a normal gamete would lead to the ...
Page 132
... chromosome 21. The obligate Down's syndrome region 21q22 is enlarged , and genes mapped to this region are indicated . The orientation of these genes with respect to each other is not known . gene ets - 2 could be linked to the ...
... chromosome 21. The obligate Down's syndrome region 21q22 is enlarged , and genes mapped to this region are indicated . The orientation of these genes with respect to each other is not known . gene ets - 2 could be linked to the ...
Page 261
... chromosome dele- tions , and ( 3 ) cytogenetic manifestations of gene am- plification.31 In addition , there may be gains and losses of whole chromosomes . In general , numerical changes are less specific . Some examples of karyo- typic ...
... chromosome dele- tions , and ( 3 ) cytogenetic manifestations of gene am- plification.31 In addition , there may be gains and losses of whole chromosomes . In general , numerical changes are less specific . Some examples of karyo- typic ...
Contents
Cellular Injury and Adaptation | 1 |
Inflammation and Repair | 39 |
Fluid and Hemodynamic Derangements | 87 |
Copyright | |
28 other sections not shown
Common terms and phrases
abnormal acid activity acute adult agents amyloid amyloidosis antibodies antigens arteries atherosclerosis bacterial blood bone cancer carcinogens carcinoma cardiac cause cellular changes chemical chromosome chronic clinical collagen complex damage death defects deficiency develop diagnosis disease disorders drugs edema effects endothelium Engl enzymes epithelial fatty fever fibroblasts fibrosis Figure fluid function gene genetic granulomas growth factor hemorrhage hepatic histologic host human immune immunologic increased induce infants infarction infection infiltration inflammation inflammatory injury interstitial intestinal intracellular involved lesions leukemia leukocytes levels lipid liver lung lymph nodes lymphocytes lysosomal macrophages malignant mechanisms mediators membrane metabolism monocytes mucosa muscle mutations necrosis necrotic neoplasms neutrophils normal occur oncogenes organs parasites pathogenesis patients phagocytic phagocytosis plasma platelet produce proliferation protein pulmonary reaction receptor renal respiratory response result risk serum skin specific syndrome synthesis tion tissue toxic tumor tumor cells ulcer usually vascular vessels viral virus viruses vitamin vitamin D