Robbins' Pathologic Basis of Disease, Volume 1Presenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
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Page 1315
SYSTEM. BONE REMODELING HEREDITARY DISORDERS Osteogenesis
Imperfecta (01) Osteopetrosis Achondroplasia Hereditary Multiple and Sporadic
Exostoses INFECTIONS Pyogenic Osteomyelitis Tuberculosis Syphilis
FRACTURES ...
SYSTEM. BONE REMODELING HEREDITARY DISORDERS Osteogenesis
Imperfecta (01) Osteopetrosis Achondroplasia Hereditary Multiple and Sporadic
Exostoses INFECTIONS Pyogenic Osteomyelitis Tuberculosis Syphilis
FRACTURES ...
Page 1317
ing unmineralized osteoid seams up to 15 //m thickness at bone-forming fronts. A
deficiency in the width and extent of the osteoid seams implies impaired
osteoblastic function and reduced bone formation and, conversely, an increase in
their ...
ing unmineralized osteoid seams up to 15 //m thickness at bone-forming fronts. A
deficiency in the width and extent of the osteoid seams implies impaired
osteoblastic function and reduced bone formation and, conversely, an increase in
their ...
Page 1330
In the second mixed phase, the resorption is closely followed by new bone
formation effected by an abundance of osteoblasts. However, the new matrix is
haphazardly laid down, mostly in the form of woven bone, some of which may be
...
In the second mixed phase, the resorption is closely followed by new bone
formation effected by an abundance of osteoblasts. However, the new matrix is
haphazardly laid down, mostly in the form of woven bone, some of which may be
...
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Contents
Cellular Injury and Adaptation l | 9 |
Inflammation and Repair | 39 |
Fluid and Hemodynamic Derangements | 87 |
Copyright | |
28 other sections not shown
Common terms and phrases
abnormal acid activity acute adult agents amyloid amyloidosis antibodies antigens arteries atherosclerosis bacterial blood bone cancer carcinogens carcinoma cardiac cause cellular changes chemical chromosome chronic clinical collagen common complex damage death defects deficiency develop diagnosis disease disorders drugs edema effects endothelial cells endothelium Engl enzymes epithelial fatty fever fibroblasts fibrosis Figure fluid focal function gene genetic granulomas growth factor hemorrhage hepatic histologic host human immune immunologic increased induce infants infarction infection infiltration inflammation inflammatory injury interstitial intestinal intracellular involved lesions leukemia leukocytes levels lipid liver lung lymph nodes lymphocytes lysosomal macrophages malignant mechanisms mediators membrane metabolism monocytes mucosa muscle mutations necrosis necrotic neoplasms neutrophils normal occur oncogenes organs parasites pathogenesis patients phagocytic phagocytosis plasma platelet produce proliferation protein pulmonary reaction receptor renal respiratory response result risk serum skin specific syndrome synthesis tion tissue toxic tumor tumor cells ulcer usually vascular vessels viral virus viruses vitamin
Bibliographic information
| Title | Robbins' Pathologic Basis of Disease, Volume 1 Robbins' pathologic basis of disease, Stanley Leonard Robbins |
| Contributors | Stanley Leonard Robbins, Ramzi S. Cotran, Vinay Kumar |
| Edition | 4 |
| Publisher | Saunders, 1989 |
| Original from | University of Minnesota |
| Digitized | 21 Jan 2010 |
| ISBN | 0721623026, 9780721623023 |
| Length | 1519 pages |
| Export Citation | BiBTeX EndNote RefMan |