Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Results 1-3 of 78
Page 148
... result from lack of tyrosinase , which is neces- sary for the biosynthesis of melanin from its precursor , tyrosine . This results in the clinical condition called albinism . If the end product is a feedback inhibitor of the enzymes ...
... result from lack of tyrosinase , which is neces- sary for the biosynthesis of melanin from its precursor , tyrosine . This results in the clinical condition called albinism . If the end product is a feedback inhibitor of the enzymes ...
Page 169
... result from chromosome breakage followed by loss or rearrangement of material . Such alterations occur spontane- ously at a low rate that is increased by exposure to envi- ronmental mutagens , such as chemicals and ionizing radia- tions ...
... result from chromosome breakage followed by loss or rearrangement of material . Such alterations occur spontane- ously at a low rate that is increased by exposure to envi- ronmental mutagens , such as chemicals and ionizing radia- tions ...
Page 727
... results in the release of mediators that can injure parenchymal cells and stimulate fibrosis . The final result is an end - stage fibrotic lung in which the alveoli are replaced by cystic spaces separated by thick bands of con- nective ...
... results in the release of mediators that can injure parenchymal cells and stimulate fibrosis . The final result is an end - stage fibrotic lung in which the alveoli are replaced by cystic spaces separated by thick bands of con- nective ...
Contents
Cell Injury and Diseases of Immunity 188 | 1 |
29 | 14 |
Neoplasia 260 | 21 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion allele amyloidosis angiogenesis antibodies antigens apoptosis associated autosomal bacterial binding cancer carcinoma cause CD4+ cell cycle cell death cell injury cellular Chapter chemokines chromosome chronic clinical coagulation collagen common complex cyclin cytokines damage defects deficiency develop disease disorders edema effects endothelial cells endothelium enzyme epithelial example extracellular extracellular matrix fibrin fibroblasts fibrosis Figure function gene genetic glycogen granules growth factors hepatic host human immune increased induced infarction infection inflammation inflammatory inhibit integrins involved kinase lesions leukocyte lipid liver lung lymphocytes lysosomal macrophages matrix mechanisms mediators membrane metabolism molecular molecules monocytes muscle mutations necrosis neoplasms neutrophils normal occur organs oxidative pathogenesis pathway patients phagocytosis plasma platelet produce proliferation protein pulmonary reactions receptor regulation release renal response result signals skin specific surface syndrome synthesis T-cell thrombi thrombosis tion toxic tumor tumor cells vascular viral virus viruses vitamin