Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 54
... mechanisms have been proposed ( Fig . 3-4 ) : Formation of endothelial gaps in venules : This is the most common mechanism of vascular leakage and is elicited by histamine , bradykinin , leukotrienes , substance P , and many other ...
... mechanisms have been proposed ( Fig . 3-4 ) : Formation of endothelial gaps in venules : This is the most common mechanism of vascular leakage and is elicited by histamine , bradykinin , leukotrienes , substance P , and many other ...
Page 214
... mechanisms of self - tolerance , it is believed that additional fail - safe mechanisms must also exist . Much interest is focused on suppressor T cells with the ability to down - regu- late the function of other autoreactive T cells ...
... mechanisms of self - tolerance , it is believed that additional fail - safe mechanisms must also exist . Much interest is focused on suppressor T cells with the ability to down - regu- late the function of other autoreactive T cells ...
Page 318
... mechanisms may also participate in tumor cell destruction by two mechanisms : activation of comple- ment and induction of ADCC by natural killer cells . IMMUNOSURVEILLANCE Given the host of possible and potential antitumor mech- anisms ...
... mechanisms may also participate in tumor cell destruction by two mechanisms : activation of comple- ment and induction of ADCC by natural killer cells . IMMUNOSURVEILLANCE Given the host of possible and potential antitumor mech- anisms ...
Contents
Cell Injury and Diseases of Immunity 188 | 1 |
29 | 14 |
Neoplasia 260 | 21 |
Copyright | |
23 other sections not shown
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Common terms and phrases
abnormal acid activation acute adhesion allele amyloidosis angiogenesis antibodies antigens apoptosis associated autosomal bacterial binding cancer carcinoma cause CD4+ cell cycle cell death cell injury cellular Chapter chemokines chromosome chronic clinical coagulation collagen common complex cyclin cytokines damage defects deficiency develop disease disorders edema effects endothelial cells endothelium enzyme epithelial example extracellular extracellular matrix fibrin fibroblasts fibrosis Figure function gene genetic glycogen granules growth factors hepatic host human immune increased induced infarction infection inflammation inflammatory inhibit integrins involved kinase lesions leukocyte lipid liver lung lymphocytes lysosomal macrophages matrix mechanisms mediators membrane metabolism molecular molecules monocytes muscle mutations necrosis neoplasms neutrophils normal occur organs oxidative pathogenesis pathway patients phagocytosis plasma platelet produce proliferation protein pulmonary reactions receptor regulation release renal response result signals skin specific surface syndrome synthesis T-cell thrombi thrombosis tion toxic tumor tumor cells vascular viral virus viruses vitamin