Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Results 1-3 of 82
Page 82
... lymphocytes , plasma cells , mast cells , and eosinophils : Lymphocytes are mobilized in both antibody - mediated and cell - mediated immune reactions and , for reasons largely unknown , in non - immune - mediated inflamma- tion .
... lymphocytes , plasma cells , mast cells , and eosinophils : Lymphocytes are mobilized in both antibody - mediated and cell - mediated immune reactions and , for reasons largely unknown , in non - immune - mediated inflamma- tion .
Page 427
... Lymphocytes < 1500 / mm3 Intermittent nausea and vomiting Petechiae , hemorrhage Maximum neutrophil and platelet depression in 2 wk Lymphocytes < 1000 / mm3 Nausea , vomiting , diarrhea Hemorrhage and infection in 1-3 wk Severe ...
... Lymphocytes < 1500 / mm3 Intermittent nausea and vomiting Petechiae , hemorrhage Maximum neutrophil and platelet depression in 2 wk Lymphocytes < 1000 / mm3 Nausea , vomiting , diarrhea Hemorrhage and infection in 1-3 wk Severe ...
Page 673
... lymphocytes with few admixed macrophages or other reactive ele- ments . In such lymphocyte - rich cases , immuno- phenotyping may be needed to exclude the lym- phocyte predominance type of HD or an unusual NHL . The mixed cellularity ...
... lymphocytes with few admixed macrophages or other reactive ele- ments . In such lymphocyte - rich cases , immuno- phenotyping may be needed to exclude the lym- phocyte predominance type of HD or an unusual NHL . The mixed cellularity ...
Contents
Cell Injury and Diseases of Immunity 188 | 1 |
29 | 14 |
Neoplasia 260 | 21 |
Copyright | |
23 other sections not shown
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Common terms and phrases
abnormal acid activation acute adhesion allele amyloidosis angiogenesis antibodies antigens apoptosis associated autosomal bacterial binding cancer carcinoma cause CD4+ cell cycle cell death cell injury cellular Chapter chemokines chromosome chronic clinical coagulation collagen common complex cyclin cytokines damage defects deficiency develop disease disorders edema effects endothelial cells endothelium enzyme epithelial example extracellular extracellular matrix fibrin fibroblasts fibrosis Figure function gene genetic glycogen granules growth factors hepatic host human immune increased induced infarction infection inflammation inflammatory inhibit integrins involved kinase lesions leukocyte lipid liver lung lymphocytes lysosomal macrophages matrix mechanisms mediators membrane metabolism molecular molecules monocytes muscle mutations necrosis neoplasms neutrophils normal occur organs oxidative pathogenesis pathway patients phagocytosis plasma platelet produce proliferation protein pulmonary reactions receptor regulation release renal response result signals skin specific surface syndrome synthesis T-cell thrombi thrombosis tion toxic tumor tumor cells vascular viral virus viruses vitamin