Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
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Page 14
... lipid is initiated , and or- ganic peroxides are formed after reacting with oxygen ( lipid peroxidation ) . This reaction is autocatalytic in that new radicals are formed from the peroxide radicals them- selves . Thus , rapid breakdown ...
... lipid is initiated , and or- ganic peroxides are formed after reacting with oxygen ( lipid peroxidation ) . This reaction is autocatalytic in that new radicals are formed from the peroxide radicals them- selves . Thus , rapid breakdown ...
Page 15
... Lipid radicals +02 LIPID PEROXIDATION Membrane damage to RER Autocatalytic spread along microsomal membrane Polysome detachment + Apoprotein synthesis Release of products of lipid peroxidation Damage to plasma membrane + Permeability to ...
... Lipid radicals +02 LIPID PEROXIDATION Membrane damage to RER Autocatalytic spread along microsomal membrane Polysome detachment + Apoprotein synthesis Release of products of lipid peroxidation Damage to plasma membrane + Permeability to ...
Page 503
... Lipid core and fibrotic layer , or multiple lipid cores and fibrotic hematoma - hemorrhage , thrombus Table 12-3 . HYPERLIPOPROTEINEMIAS AND THEIR GENETIC BASIS LDL ,. hemorrhage ) . Thrombi may partially or com- pletely occlude the ...
... Lipid core and fibrotic layer , or multiple lipid cores and fibrotic hematoma - hemorrhage , thrombus Table 12-3 . HYPERLIPOPROTEINEMIAS AND THEIR GENETIC BASIS LDL ,. hemorrhage ) . Thrombi may partially or com- pletely occlude the ...
Contents
Cell Injury and Diseases of Immunity 188 | 1 |
29 | 14 |
Neoplasia 260 | 21 |
Copyright | |
23 other sections not shown
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Common terms and phrases
abnormal acid activation acute adhesion allele amyloidosis angiogenesis antibodies antigens apoptosis associated autosomal bacterial binding cancer carcinoma cause CD4+ cell cycle cell death cell injury cellular Chapter chemokines chromosome chronic clinical coagulation collagen common complex cyclin cytokines damage defects deficiency develop disease disorders edema effects endothelial cells endothelium enzyme epithelial example extracellular extracellular matrix fibrin fibroblasts fibrosis Figure function gene genetic glycogen granules growth factors hepatic host human immune increased induced infarction infection inflammation inflammatory inhibit integrins involved kinase lesions leukocyte lipid liver lung lymphocytes lysosomal macrophages matrix mechanisms mediators membrane metabolism molecular molecules monocytes muscle mutations necrosis neoplasms neutrophils normal occur organs oxidative pathogenesis pathway patients phagocytosis plasma platelet produce proliferation protein pulmonary reactions receptor regulation release renal response result signals skin specific surface syndrome synthesis T-cell thrombi thrombosis tion toxic tumor tumor cells vascular viral virus viruses vitamin