Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
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Page 100
... binding domains . Collagen Fibronectin Laminin fibers Laminin fibers EXTRACELLULAR MATRIX Fibronectin Collagen. walls ... binding motif RGD1 COOH NH2 Heparan- Fibrin- binding binding binding domains domains domains NH2 77 SS Collagen ...
... binding domains . Collagen Fibronectin Laminin fibers Laminin fibers EXTRACELLULAR MATRIX Fibronectin Collagen. walls ... binding motif RGD1 COOH NH2 Heparan- Fibrin- binding binding binding domains domains domains NH2 77 SS Collagen ...
Page 102
... binds poorly to the FGF recep- tor . Binding of FGF to the heparan sulfate chains like those on syndecan allows FGF to bind efficiently to its receptor . Binding of FGF to heparan sulfate in the ECM protects FGF and forms a reservoir of ...
... binds poorly to the FGF recep- tor . Binding of FGF to the heparan sulfate chains like those on syndecan allows FGF to bind efficiently to its receptor . Binding of FGF to heparan sulfate in the ECM protects FGF and forms a reservoir of ...
Page 194
... bind to the antigen - binding cleft of newly synthe- sized class I heavy chains , which then associate with B2- microglobulin to form a stable trimer that is transported to the cell surface for presentation to the CD8 + cytotoxic T ...
... bind to the antigen - binding cleft of newly synthe- sized class I heavy chains , which then associate with B2- microglobulin to form a stable trimer that is transported to the cell surface for presentation to the CD8 + cytotoxic T ...
Contents
Cell Injury and Diseases of Immunity 188 | 1 |
29 | 14 |
Neoplasia 260 | 21 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion allele amyloidosis angiogenesis antibodies antigens apoptosis associated autosomal bacterial binding cancer carcinoma cause CD4+ cell cycle cell death cell injury cellular Chapter chemokines chromosome chronic clinical coagulation collagen common complex cyclin cytokines damage defects deficiency develop disease disorders edema effects endothelial cells endothelium enzyme epithelial example extracellular extracellular matrix fibrin fibroblasts fibrosis Figure function gene genetic glycogen granules growth factors hepatic host human immune increased induced infarction infection inflammation inflammatory inhibit integrins involved kinase lesions leukocyte lipid liver lung lymphocytes lysosomal macrophages matrix mechanisms mediators membrane metabolism molecular molecules monocytes muscle mutations necrosis neoplasms neutrophils normal occur organs oxidative pathogenesis pathway patients phagocytosis plasma platelet produce proliferation protein pulmonary reactions receptor regulation release renal response result signals skin specific surface syndrome synthesis T-cell thrombi thrombosis tion toxic tumor tumor cells vascular viral virus viruses vitamin