Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 340
... virus and fusion with endoso- mal membranes . Within the cell , the virus uncoats , separat- ing its genome from its structural components and losing its infectivity . Viruses then replicate with use of enzymes , which are distinct for each ...
... virus and fusion with endoso- mal membranes . Within the cell , the virus uncoats , separat- ing its genome from its structural components and losing its infectivity . Viruses then replicate with use of enzymes , which are distinct for each ...
Page 347
... viruses ( e.g. , influenza viruses ) possess hemagglutinins , which at- tach to epithelial surface carbohydrates and thus prevent mucociliary clearance . Others also have neuraminidases , which degrade respiratory mucus and thereby ...
... viruses ( e.g. , influenza viruses ) possess hemagglutinins , which at- tach to epithelial surface carbohydrates and thus prevent mucociliary clearance . Others also have neuraminidases , which degrade respiratory mucus and thereby ...
Page 348
... VIRUSES Influenza viruses are larger and more complex than rhi- noviruses . The genome of influenza virus is composed of eight helices of single - stranded RNA , each encoding a single gene and each bound by a nucleoprotein that ...
... VIRUSES Influenza viruses are larger and more complex than rhi- noviruses . The genome of influenza virus is composed of eight helices of single - stranded RNA , each encoding a single gene and each bound by a nucleoprotein that ...
Contents
Acute and Chronic Inflammation | 50 |
Cellular Growth Fibrosis | 89 |
Hemodynamic Disorders Thrombosis | 113 |
Copyright | |
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abnormal acid activation acute adhesion allele amyloidosis antibodies antigens apoptosis arteries associated atherosclerosis bacterial benign binding blood cancer carcinoma cause CD4+ cell cycle cell death cellular Chapter chromosome chronic clinical collagen colon common complex cyclin cytokines damage defects deficiency develop disease disorders DNA repair edema effects endothelial cells endothelium enzymes epithelial example fibrosis Figure function gene genetic growth factors host human hypertension immune increased induced infection inflammation inflammatory integrins intracellular involved kinase lesions leukemia leukocytes lipid liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY mutations necrosis neoplasms neutrophils normal occur oncogenes organs oxidative pathogenesis Pathology pathway patients plasma platelet produce proliferation protein protooncogenes pulmonary reaction receptor regulate renal respiratory response result risk signals skin smooth muscle surface syndrome synthesis T-cell tion tissue toxic transcription tumor tumor cells vascular vasculitis vessels viral virus viruses vitamin