Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Results 1-3 of 83
Page 301
... cell Death Nonproliferative pool Figure 8-38 Go Go Differentiation Schematic representation of tumor growth . As the cell population ex- pands , a progressively higher percentage of tumor cells leaves the replica- tive pool by reversion ...
... cell Death Nonproliferative pool Figure 8-38 Go Go Differentiation Schematic representation of tumor growth . As the cell population ex- pands , a progressively higher percentage of tumor cells leaves the replica- tive pool by reversion ...
Page 305
... TUMOR CELLS Once in circulation , tumor cells are particularly vulnera- ble to destruction by natural and adaptive immune de- fenses . The details of tumor immunity are considered later . Suffice it to say that natural killer cells seem ...
... TUMOR CELLS Once in circulation , tumor cells are particularly vulnera- ble to destruction by natural and adaptive immune de- fenses . The details of tumor immunity are considered later . Suffice it to say that natural killer cells seem ...
Page 318
... cells is depicted in Figure 8-45 . Thus , natural killer cells may provide the first line of defense against many ... tumor cells that escape T - cell recognition may succumb to natural killer cells . Adoptive immuno- therapy with in ...
... cells is depicted in Figure 8-45 . Thus , natural killer cells may provide the first line of defense against many ... tumor cells that escape T - cell recognition may succumb to natural killer cells . Adoptive immuno- therapy with in ...
Contents
Acute and Chronic Inflammation | 50 |
Cellular Growth Fibrosis | 89 |
Hemodynamic Disorders Thrombosis | 113 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion allele amyloidosis antibodies antigens apoptosis arteries associated atherosclerosis bacterial benign binding blood cancer carcinoma cause CD4+ cell cycle cell death cellular Chapter chromosome chronic clinical collagen colon common complex cyclin cytokines damage defects deficiency develop disease disorders DNA repair edema effects endothelial cells endothelium enzymes epithelial example fibrosis Figure function gene genetic growth factors host human hypertension immune increased induced infection inflammation inflammatory integrins intracellular involved kinase lesions leukemia leukocytes lipid liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY mutations necrosis neoplasms neutrophils normal occur oncogenes organs oxidative pathogenesis Pathology pathway patients plasma platelet produce proliferation protein protooncogenes pulmonary reaction receptor regulate renal respiratory response result risk signals skin smooth muscle surface syndrome synthesis T-cell tion tissue toxic transcription tumor tumor cells vascular vasculitis vessels viral virus viruses vitamin