Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
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Page 118
... Release of : t - PA ( fibrinolysis ) ⚫ thrombomodulin ( blocks coagulation cascade ) Trapped neutrophil Trapped red blood cells INTRINSIC PATHWAY XII ( Hageman Factor ) HMWK collagen Fibrin. bruise ) or may accumulate sufficient blood ...
... Release of : t - PA ( fibrinolysis ) ⚫ thrombomodulin ( blocks coagulation cascade ) Trapped neutrophil Trapped red blood cells INTRINSIC PATHWAY XII ( Hageman Factor ) HMWK collagen Fibrin. bruise ) or may accumulate sufficient blood ...
Page 227
... release of platelet factors ( e.g. , PDGF , TGF - B ) that trigger periadventitial fi- brosis . Activated or injured endothelial cells themselves may release PDGF and factors chemotactic for fibroblasts . Eventually , widespread ...
... release of platelet factors ( e.g. , PDGF , TGF - B ) that trigger periadventitial fi- brosis . Activated or injured endothelial cells themselves may release PDGF and factors chemotactic for fibroblasts . Eventually , widespread ...
Page 1255
... release of crystals into the synovial fluid followed by a cascade of events . The released crystals are chemotactic to leukocytes and also activate complement , with the generation of C3a and C5a leading to the accumu- lation of ...
... release of crystals into the synovial fluid followed by a cascade of events . The released crystals are chemotactic to leukocytes and also activate complement , with the generation of C3a and C5a leading to the accumu- lation of ...
Contents
Acute and Chronic Inflammation | 50 |
Cellular Growth Fibrosis | 89 |
Hemodynamic Disorders Thrombosis | 113 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion allele amyloidosis antibodies antigens apoptosis arteries associated atherosclerosis bacterial benign binding blood cancer carcinoma cause CD4+ cell cycle cell death cellular Chapter chromosome chronic clinical collagen colon common complex cyclin cytokines damage defects deficiency develop disease disorders DNA repair edema effects endothelial cells endothelium enzymes epithelial example fibrosis Figure function gene genetic growth factors host human hypertension immune increased induced infection inflammation inflammatory integrins intracellular involved kinase lesions leukemia leukocytes lipid liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY mutations necrosis neoplasms neutrophils normal occur oncogenes organs oxidative pathogenesis Pathology pathway patients plasma platelet produce proliferation protein protooncogenes pulmonary reaction receptor regulate renal respiratory response result risk signals skin smooth muscle surface syndrome synthesis T-cell tion tissue toxic transcription tumor tumor cells vascular vasculitis vessels viral virus viruses vitamin