Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
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Page 6
... permeability . Early loss of selec- tive membrane permeability leading ultimately to overt membrane damage is a consistent feature of all forms of cell injury . Such defects may be the result of a series of events involving ATP ...
... permeability . Early loss of selec- tive membrane permeability leading ultimately to overt membrane damage is a consistent feature of all forms of cell injury . Such defects may be the result of a series of events involving ATP ...
Page 53
... permeability of small blood vessels in the area of injury . In contrast , a transudate is a fluid with low protein content ( most of which is albumin ) and a specific gravity of less than 1.012 . It is essentially an ultrafiltrate of ...
... permeability of small blood vessels in the area of injury . In contrast , a transudate is a fluid with low protein content ( most of which is albumin ) and a specific gravity of less than 1.012 . It is essentially an ultrafiltrate of ...
Page 87
... permeability to macromolecules by vascular permeability fac- tor , histamine and serotonin . J Exp Med 183 : 1981 , 1996 . 10. Lampugnani MG , Dejana E : Interendothelial junctions : structure , sig- nalling and functional roles . Curr ...
... permeability to macromolecules by vascular permeability fac- tor , histamine and serotonin . J Exp Med 183 : 1981 , 1996 . 10. Lampugnani MG , Dejana E : Interendothelial junctions : structure , sig- nalling and functional roles . Curr ...
Contents
Acute and Chronic Inflammation | 50 |
Cellular Growth Fibrosis | 89 |
Hemodynamic Disorders Thrombosis | 113 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion allele amyloidosis antibodies antigens apoptosis arteries associated atherosclerosis bacterial benign binding blood cancer carcinoma cause CD4+ cell cycle cell death cellular Chapter chromosome chronic clinical collagen colon common complex cyclin cytokines damage defects deficiency develop disease disorders DNA repair edema effects endothelial cells endothelium enzymes epithelial example fibrosis Figure function gene genetic growth factors host human hypertension immune increased induced infection inflammation inflammatory integrins intracellular involved kinase lesions leukemia leukocytes lipid liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY mutations necrosis neoplasms neutrophils normal occur oncogenes organs oxidative pathogenesis Pathology pathway patients plasma platelet produce proliferation protein protooncogenes pulmonary reaction receptor regulate renal respiratory response result risk signals skin smooth muscle surface syndrome synthesis T-cell tion tissue toxic transcription tumor tumor cells vascular vasculitis vessels viral virus viruses vitamin