Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 556
... myocardium ( with blood flow of 10 % or less of normal ) .27 Microvascular injury follows . This time frame is summarized in Table 13-3 . Classic acute MI with extensive damage occurs when the perfusion of the myocardium is reduced ...
... myocardium ( with blood flow of 10 % or less of normal ) .27 Microvascular injury follows . This time frame is summarized in Table 13-3 . Classic acute MI with extensive damage occurs when the perfusion of the myocardium is reduced ...
Page 559
... myocardium and thereby limit infarct size , with con- sequent improvement in both short - term and long - term function and survival.31 As discussed previously , loss of myocardial viability in infarction is progressive , occurring over ...
... myocardium and thereby limit infarct size , with con- sequent improvement in both short - term and long - term function and survival.31 As discussed previously , loss of myocardial viability in infarction is progressive , occurring over ...
Page 561
... myocardium modified by reperfu- sion . A , Large , densely hemorrhagic , anterior wall acute myocardial in- farction from a patient with left anterior descending ( LAD ) artery throm- bus treated with streptokinase intracoronary ...
... myocardium modified by reperfu- sion . A , Large , densely hemorrhagic , anterior wall acute myocardial in- farction from a patient with left anterior descending ( LAD ) artery throm- bus treated with streptokinase intracoronary ...
Contents
Acute and Chronic Inflammation | 50 |
Cellular Growth Fibrosis | 89 |
Hemodynamic Disorders Thrombosis | 113 |
Copyright | |
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abnormal acid activation acute adhesion allele amyloidosis antibodies antigens apoptosis arteries associated atherosclerosis bacterial benign binding blood cancer carcinoma cause CD4+ cell cycle cell death cellular Chapter chromosome chronic clinical collagen colon common complex cyclin cytokines damage defects deficiency develop disease disorders DNA repair edema effects endothelial cells endothelium enzymes epithelial example fibrosis Figure function gene genetic growth factors host human hypertension immune increased induced infection inflammation inflammatory integrins intracellular involved kinase lesions leukemia leukocytes lipid liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY mutations necrosis neoplasms neutrophils normal occur oncogenes organs oxidative pathogenesis Pathology pathway patients plasma platelet produce proliferation protein protooncogenes pulmonary reaction receptor regulate renal respiratory response result risk signals skin smooth muscle surface syndrome synthesis T-cell tion tissue toxic transcription tumor tumor cells vascular vasculitis vessels viral virus viruses vitamin