Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
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Page 55
... Leukocyte - mediated endothelial injury : Leukocytes ad- here to endothelium relatively early in inflammation . As seen subsequently , such leukocytes may be activated in the process , releasing toxic oxygen species and proteo- lytic ...
... Leukocyte - mediated endothelial injury : Leukocytes ad- here to endothelium relatively early in inflammation . As seen subsequently , such leukocytes may be activated in the process , releasing toxic oxygen species and proteo- lytic ...
Page 57
... leukocyte accumulation is called margina- tion . Subsequently , individual and then rows of leukocytes tumble slowly along the endothelium and adhere transiently ( a process called rolling ) , finally coming to rest at some point where ...
... leukocyte accumulation is called margina- tion . Subsequently , individual and then rows of leukocytes tumble slowly along the endothelium and adhere transiently ( a process called rolling ) , finally coming to rest at some point where ...
Page 61
... Leukocyte diapedesis , similar to increased vascular permeability , occurs predominantly in the venules ( except in the lungs , where it also occurs in capil- laries ) . After traversing the endothelium , leukocytes are tran- siently ...
... Leukocyte diapedesis , similar to increased vascular permeability , occurs predominantly in the venules ( except in the lungs , where it also occurs in capil- laries ) . After traversing the endothelium , leukocytes are tran- siently ...
Contents
Acute and Chronic Inflammation | 50 |
Cellular Growth Fibrosis | 89 |
Hemodynamic Disorders Thrombosis | 113 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion allele amyloidosis antibodies antigens apoptosis arteries associated atherosclerosis bacterial benign binding blood cancer carcinoma cause CD4+ cell cycle cell death cellular Chapter chromosome chronic clinical collagen colon common complex cyclin cytokines damage defects deficiency develop disease disorders DNA repair edema effects endothelial cells endothelium enzymes epithelial example fibrosis Figure function gene genetic growth factors host human hypertension immune increased induced infection inflammation inflammatory integrins intracellular involved kinase lesions leukemia leukocytes lipid liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY mutations necrosis neoplasms neutrophils normal occur oncogenes organs oxidative pathogenesis Pathology pathway patients plasma platelet produce proliferation protein protooncogenes pulmonary reaction receptor regulate renal respiratory response result risk signals skin smooth muscle surface syndrome synthesis T-cell tion tissue toxic transcription tumor tumor cells vascular vasculitis vessels viral virus viruses vitamin