Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 256
Voscular involvement ond Kupffer cell depositions ore frequent. Normal liver
function is usually preserved despite sometimes quite severe involvement of the
liver. Heart. Amyloidosis of the heart moy occur in ony form of systemic
dmyloidosis, ...
Voscular involvement ond Kupffer cell depositions ore frequent. Normal liver
function is usually preserved despite sometimes quite severe involvement of the
liver. Heart. Amyloidosis of the heart moy occur in ony form of systemic
dmyloidosis, ...
Page 735
Pothologic involvement of virtually every organ in the body has been cited of one
time Or Cnother, The lungs ore common sites of involvement. Mocroscopically,
there is usually no demonstroble alteration, although at times the coolescence of
...
Pothologic involvement of virtually every organ in the body has been cited of one
time Or Cnother, The lungs ore common sites of involvement. Mocroscopically,
there is usually no demonstroble alteration, although at times the coolescence of
...
Page 1252
JRA is classified into oligoarticular (<5 joints involved), polyarticular (>5 joints
involved), and systemic variants. JRA differs from RA in adults in the following
points: (1) Oligoarthritis is more common, (2) systemic onset is more frequent, ...
JRA is classified into oligoarticular (<5 joints involved), polyarticular (>5 joints
involved), and systemic variants. JRA differs from RA in adults in the following
points: (1) Oligoarthritis is more common, (2) systemic onset is more frequent, ...
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Contents
Z S 6 S GENERAL PATHOLOGY Genetic Disorders 139 | 1 |
Neoplasia 260 | 21 |
Adaptations | 31 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria binding blood cancer carcinoma caspase cause CD4+ cell cycle cell death cell injury cellular Chapter chemical chemokines chromosome chronic clinical collagen common complex couse cyclin cytokines cytoplasm damage defects deficiency develop disease disorders edema effects endothelial cells endothelium enzymes epithelial fibrin fibroblasts fibrosis Figure function gene genetic granules growth factors host immune increased induced infection inflammation inflammatory inhibit integrins intracellular involved kinase lesions leukocyte lipid liver lung lymphocytes lysosomal macrophages matrix mechanisms mediators membrane metabolism mitochondrial molecular molecules MORPHOLOGY muscle mutations necrosis neutrophils normal occur organs oxidative pathogenesis Pathology pathway patients permeability phagocytosis phospholipids plasma platelet produced proliferation proteases protein reactions receptors regulation release renal response result signals skin specific surface syndrome synthesis T-cell thrombi tion tissue toxic tumor tumor cells vascular vessels viral virus vitamin
References to this book
Bibliographic information
| Title | Robbins Pathologic Basis of Disease MD consult Robbins Pathology Series Saunders W.B |
| Author | Ramzi S. Cotran |
| Editor | Vinay Kumar |
| Contributors | Tucker Collins, Stanley Leonard Robbins |
| Edition | 6, illustrated |
| Publisher | Saunders, 1999 |
| Original from | the University of Michigan |
| Digitized | 4 Aug 2008 |
| ISBN | 072167335X, 9780721673356 |
| Length | 1425 pages |
| Subjects | › Medical / Pathology |
| Export Citation | BiBTeX EndNote RefMan |