Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 511
... hypertension . About 90 to 95 % of hypertension is idiopathic and appar- ently primary ( essential hypertension ) . Of the remaining 5 to 10 % , most is secondary to renal disease or , less often , to narrowing of the renal artery ...
... hypertension . About 90 to 95 % of hypertension is idiopathic and appar- ently primary ( essential hypertension ) . Of the remaining 5 to 10 % , most is secondary to renal disease or , less often , to narrowing of the renal artery ...
Page 513
... hypertension . Genetic Factors . It is now thought that essential hyper- tension results from an interaction of genetic and environ- mental factors that affect cardiac output , peripheral resist- ance , or both . A generally accepted ...
... hypertension . Genetic Factors . It is now thought that essential hyper- tension results from an interaction of genetic and environ- mental factors that affect cardiac output , peripheral resist- ance , or both . A generally accepted ...
Page 705
... hypertension occurs in patients with mitral stenosis , for example , because of an increase in left atrial pres- sure that leads to an increase in pulmonary venous pres- sure and , consequently , to an increase in pulmonary artery ...
... hypertension occurs in patients with mitral stenosis , for example , because of an increase in left atrial pres- sure that leads to an increase in pulmonary venous pres- sure and , consequently , to an increase in pulmonary artery ...
Contents
Acute and Chronic Inflammation | 50 |
Cellular Growth Fibrosis | 89 |
Hemodynamic Disorders Thrombosis | 113 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion allele amyloidosis antibodies antigens apoptosis arteries associated atherosclerosis bacterial benign binding blood cancer carcinoma cause CD4+ cell cycle cell death cellular Chapter chromosome chronic clinical collagen colon common complex cyclin cytokines damage defects deficiency develop disease disorders DNA repair edema effects endothelial cells endothelium enzymes epithelial example fibrosis Figure function gene genetic growth factors host human hypertension immune increased induced infection inflammation inflammatory integrins intracellular involved kinase lesions leukemia leukocytes lipid liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY mutations necrosis neoplasms neutrophils normal occur oncogenes organs oxidative pathogenesis Pathology pathway patients plasma platelet produce proliferation protein protooncogenes pulmonary reaction receptor regulate renal respiratory response result risk signals skin smooth muscle surface syndrome synthesis T-cell tion tissue toxic transcription tumor tumor cells vascular vasculitis vessels viral virus viruses vitamin