Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 2
Nevertheless, the functions of the encoded proteins and how mutations induce
disease are often still obscure. Thus, the study of pathogenesis has never been
more exciting scientifically or more relevant to the development of new therapies.
Nevertheless, the functions of the encoded proteins and how mutations induce
disease are often still obscure. Thus, the study of pathogenesis has never been
more exciting scientifically or more relevant to the development of new therapies.
Page 145
In this instance, the mutant allele is called dominant negative because it impairs
the function of a normal allele. This effect is illustrated by some forms of
osteogenesis imperfecta, characterized by marked deficiency of collagen and
severe ...
In this instance, the mutant allele is called dominant negative because it impairs
the function of a normal allele. This effect is illustrated by some forms of
osteogenesis imperfecta, characterized by marked deficiency of collagen and
severe ...
Page 179
In all cases, the mutations impair gene function by an expansion of the repeats. In
general, above a certain threshold, which varies in different conditions, the
expansions are unstable; thus, as illustrated with the fragile X syndrome, ...
In all cases, the mutations impair gene function by an expansion of the repeats. In
general, above a certain threshold, which varies in different conditions, the
expansions are unstable; thus, as illustrated with the fragile X syndrome, ...
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Contents
Z S 6 S GENERAL PATHOLOGY Genetic Disorders 139 | 1 |
Neoplasia 260 | 21 |
Adaptations | 31 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria binding blood cancer carcinoma caspase cause CD4+ cell cycle cell death cell injury cellular Chapter chemical chemokines chromosome chronic clinical collagen common complex couse cyclin cytokines cytoplasm damage defects deficiency develop disease disorders edema effects endothelial cells endothelium enzymes epithelial fibrin fibroblasts fibrosis Figure function gene genetic granules growth factors host immune increased induced infection inflammation inflammatory inhibit integrins intracellular involved kinase lesions leukocyte lipid liver lung lymphocytes lysosomal macrophages matrix mechanisms mediators membrane metabolism mitochondrial molecular molecules MORPHOLOGY muscle mutations necrosis neutrophils normal occur organs oxidative pathogenesis Pathology pathway patients permeability phagocytosis phospholipids plasma platelet produced proliferation proteases protein reactions receptors regulation release renal response result signals skin specific surface syndrome synthesis T-cell thrombi tion tissue toxic tumor tumor cells vascular vessels viral virus vitamin
References to this book
Bibliographic information
| Title | Robbins Pathologic Basis of Disease MD consult Robbins Pathology Series Saunders W.B |
| Author | Ramzi S. Cotran |
| Editor | Vinay Kumar |
| Contributors | Tucker Collins, Stanley Leonard Robbins |
| Edition | 6, illustrated |
| Publisher | Saunders, 1999 |
| Original from | the University of Michigan |
| Digitized | 4 Aug 2008 |
| ISBN | 072167335X, 9780721673356 |
| Length | 1425 pages |
| Subjects | › Medical / Pathology |
| Export Citation | BiBTeX EndNote RefMan |