Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 145
... common loss of function mutations affect regulatory proteins and subunits of mulit- meric structural proteins , the latter acting through a domi- nant negative effect . Gain of function mutations are less common ; they endow normal ...
... common loss of function mutations affect regulatory proteins and subunits of mulit- meric structural proteins , the latter acting through a domi- nant negative effect . Gain of function mutations are less common ; they endow normal ...
Page 235
... common clinical manifestations , the underlying defects are quite different and in many cases unknown . The so - called classic form , initially described in Swiss infants and believed to result from a defect in the common lymphoid stem ...
... common clinical manifestations , the underlying defects are quite different and in many cases unknown . The so - called classic form , initially described in Swiss infants and believed to result from a defect in the common lymphoid stem ...
Page 1351
... common cytogenetic abnormality is loss of chromosome 22 , especially the long arm ( 22q ) . The deletions include the region at 22q12 that harbors the NF2 gene ( see later ) . Indeed , 50 % to 60 % of meningiomas not associated with ...
... common cytogenetic abnormality is loss of chromosome 22 , especially the long arm ( 22q ) . The deletions include the region at 22q12 that harbors the NF2 gene ( see later ) . Indeed , 50 % to 60 % of meningiomas not associated with ...
Contents
Acute and Chronic Inflammation | 50 |
Cellular Growth Fibrosis | 89 |
Hemodynamic Disorders Thrombosis | 113 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion allele amyloidosis antibodies antigens apoptosis arteries associated atherosclerosis bacterial benign binding blood cancer carcinoma cause CD4+ cell cycle cell death cellular Chapter chromosome chronic clinical collagen colon common complex cyclin cytokines damage defects deficiency develop disease disorders DNA repair edema effects endothelial cells endothelium enzymes epithelial example fibrosis Figure function gene genetic growth factors host human hypertension immune increased induced infection inflammation inflammatory integrins intracellular involved kinase lesions leukemia leukocytes lipid liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY mutations necrosis neoplasms neutrophils normal occur oncogenes organs oxidative pathogenesis Pathology pathway patients plasma platelet produce proliferation protein protooncogenes pulmonary reaction receptor regulate renal respiratory response result risk signals skin smooth muscle surface syndrome synthesis T-cell tion tissue toxic transcription tumor tumor cells vascular vasculitis vessels viral virus viruses vitamin