Robbins & Cotran Pathologic Basis of Disease E-BookOne of the best-selling medical textbooks of all time, Robbins and Cotran Pathologic Basis of Disease is the one book that nearly all medical students purchase, and is also widely used by physicians worldwide. A "who's who" of pathology experts delivers the most dependable, current, and complete coverage of today's essential pathology knowledge. At the same time, masterful editing and a practical organization make mastering every concept remarkably easy. The result remains the ideal source for an optimal understanding of pathology.Offers the most authoritative and comprehensive, yet readable coverage available in any pathology textbook, making it ideal for USMLE or specialty board preparation as well as for course work.Delivers a state-of-the-art understanding of the pathologic basis of disease through completely updated coverage, including the latest cellular and molecular biology. Demonstrates every concept visually with over 1,600 full-color photomicrographs and conceptual diagrams - many revised for even better quality. Facilitates learning with an outstanding full-color, highly user-friendly design. |
Other editions - View all
Robbins and Cotran Pathologic Basis of Disease Stanley Leonard Robbins,Vinay Kumar,Ramzi S. Cotran No preview available - 2010 |
Robbins and Cotran Pathologic Basis of Disease Stanley Leonard Robbins,Vinay Kumar No preview available - 2010 |
Common terms and phrases
abnormalities acid activation acute addition agents antibodies antigens apoptosis arteries associated bacteria become bind blood body bone called cancer carcinoma cardiac cause cells cellular changes Chapter chromosome chronic clinical common complex contain cytokines cytoplasm damage death decreased defects deficiency deposition described differentiation discussed disease disorders effects endothelial cells enzymes example expression factors fibrosis FIGURE formation function genes genetic growth factors heart host human immune important increased individuals induce infection inflammation inflammatory initial injury involved known lead lesions leukocytes levels liver loss lung lymphocytes macrophages major mechanisms mediators membrane microbes molecular molecules muscle mutations myocardial infarction necrosis normal occur organs pathway patients plasma present produce progressive proliferation proteins pulmonary reactions receptors reduced regulation release response result risk role severe signaling skin specific stimulate surface syndrome Table tion tissue tumor usually vascular vessels viral virus