Curares and Curarisation: Post Graduate Course XIth International Meeting of Anaesthesiology and Resuscitation, 1979C. Conseiller |
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Page 35
... respiratory alkalosis . c ) Gallamine As far as gallamine is concerned , the different authors agree that the amplitude and duration of the neuromuscular block are increased by respiratory and metabolic alkalosis and decreased by ...
... respiratory alkalosis . c ) Gallamine As far as gallamine is concerned , the different authors agree that the amplitude and duration of the neuromuscular block are increased by respiratory and metabolic alkalosis and decreased by ...
Page 38
... Respiratory alkalosis and metabolic acidosis do not modify the latency of the inversion by neostigmine of the block induced by d - tubocurarine . Respiratory alkalosis and metabolic acidosis have no effect on the block inversion and the ...
... Respiratory alkalosis and metabolic acidosis do not modify the latency of the inversion by neostigmine of the block induced by d - tubocurarine . Respiratory alkalosis and metabolic acidosis have no effect on the block inversion and the ...
Page 39
... respiratory alkalosis and shortened by respiratory acidosis . As far as the block induced by pancuronium is concerned , respiratory irregularities do not significantly modify the duration of action of neostigmine , which is shortened in ...
... respiratory alkalosis and shortened by respiratory acidosis . As far as the block induced by pancuronium is concerned , respiratory irregularities do not significantly modify the duration of action of neostigmine , which is shortened in ...
Contents
WASER P G and SCHOENENBERGER E The structure | 7 |
S Mechanisms of action of muscle relaxants | 21 |
NIVOCHE Y Pharmacologic factors modifying the function | 53 |
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acetylcholine acting activity administration agents Anaesth anesthesia Anesthesiology anesthetics antagonism anticholinesterase appears arterial associated authors blockade blood Brit calcium cardiac cardiovascular cause cent changes clinical compared compounds concentration contraction d-tubocurarine decrease depends depolarizing depression distribution dose drug duration of action effects Eger elimination fact factors failure fazadinium function gallamine given half-life halothane heart rate hemodynamic important increase indication induced infants inhibition initial injection interaction intubation Katz leads less Marshall measured mechanical membrane metabolic mg/kg Miller minutes modifications molecule monitoring motor end-plate muscle relaxants muscular myorelaxants myosin necessary neostigmine nerve nervous neuromuscular block neuromuscular junction non-depolarizing curares normal observed pancuronium pancuronium bromide patients pharmacokinetic Pharmacol pharmacologic placenta plasma possible potential present pressure presynaptic prolonged protein receptors recorded recovery reduced release renal reported respect respiratory response seems sensitive stimulation succinylcholine Table transmission values ventilation