Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
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Page 15
... toxic metabolites accumulate in the cell , destroy nucleophilic macromolecules , and cova- lently bind proteins and nucleic acids . The decrease in GSH concentration , coupled with covalent binding of toxic metabolites , increases drug ...
... toxic metabolites accumulate in the cell , destroy nucleophilic macromolecules , and cova- lently bind proteins and nucleic acids . The decrease in GSH concentration , coupled with covalent binding of toxic metabolites , increases drug ...
Page 405
... toxic than the parent chemical . One or more of these products then interacts with the target macromolecule , resulting in a toxic effect . The site of toxicity is frequently the site where metabolism or excretion of toxic metabolites ...
... toxic than the parent chemical . One or more of these products then interacts with the target macromolecule , resulting in a toxic effect . The site of toxicity is frequently the site where metabolism or excretion of toxic metabolites ...
Page 422
... toxic . Acute toxicity of insecticides for mammals ranges from low to high . Fumigants used to eliminate insects from enclosed spaces and rodenticides are highly toxic . Whereas the acute toxicity of agricultural pes- ticides is well ...
... toxic . Acute toxicity of insecticides for mammals ranges from low to high . Fumigants used to eliminate insects from enclosed spaces and rodenticides are highly toxic . Whereas the acute toxicity of agricultural pes- ticides is well ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
18 other sections not shown
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin