Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 252
... serum called SAA ( serum amyloid - associated ) protein that is synthesized in the liver and circulates in association with the HDL3 subclass of lipoproteins . Several other biochemi- cally distinct proteins have been found in amyloid ...
... serum called SAA ( serum amyloid - associated ) protein that is synthesized in the liver and circulates in association with the HDL3 subclass of lipoproteins . Several other biochemi- cally distinct proteins have been found in amyloid ...
Page 665
... serum and more than 6 mg of Bence Jones proteins per dl of urine . The most common serum monoclonal Ig ( M protein ) is IgG , which is found in 55 % of patients . An Normal serum 2 SP GAM K λ + in some cases constitute most of the ...
... serum and more than 6 mg of Bence Jones proteins per dl of urine . The most common serum monoclonal Ig ( M protein ) is IgG , which is found in 55 % of patients . An Normal serum 2 SP GAM K λ + in some cases constitute most of the ...
Page 859
... Serum markers HBeAg HBV - DNA HBsAg Serum transaminases IgM - anti - HBc ..IgG- anti - HBs . Anti - HBe B IgG- anti - HBc 4-26 weeks ( average 8 ) 4-12 weeks Months to Years HBeAg , HBV DNA , and DNA polymerase appear in the serum soon ...
... Serum markers HBeAg HBV - DNA HBsAg Serum transaminases IgM - anti - HBc ..IgG- anti - HBs . Anti - HBe B IgG- anti - HBc 4-26 weeks ( average 8 ) 4-12 weeks Months to Years HBeAg , HBV DNA , and DNA polymerase appear in the serum soon ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
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abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin