Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 169
... result from chromosome breakage followed by loss or rearrangement of material . Such alterations occur spontane- ously at a low rate that is increased by exposure to envi- ronmental mutagens , such as chemicals and ionizing radia- tions ...
... result from chromosome breakage followed by loss or rearrangement of material . Such alterations occur spontane- ously at a low rate that is increased by exposure to envi- ronmental mutagens , such as chemicals and ionizing radia- tions ...
Page 936
... result of either diseases directly affecting tubular structure ( e.g. , medul- lary cystic disease ) or defects in specific tubular func- tions . The latter can be inherited ( e.g. , familial nephro- genic diabetes , cystinuria , renal ...
... result of either diseases directly affecting tubular structure ( e.g. , medul- lary cystic disease ) or defects in specific tubular func- tions . The latter can be inherited ( e.g. , familial nephro- genic diabetes , cystinuria , renal ...
Page 1133
... result of an infiltrative disorder . A large resection of the gland ( total thyroidec- tomy ) for the treatment of hyperthyroidism or excision of a primary neoplasm is a common cause of hypothyroidism . The gland may also be ablated by ...
... result of an infiltrative disorder . A large resection of the gland ( total thyroidec- tomy ) for the treatment of hyperthyroidism or excision of a primary neoplasm is a common cause of hypothyroidism . The gland may also be ablated by ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
18 other sections not shown
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin