Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 67
... pathway , or by the alternative pathway itself ( see text ) . Both pathways converge and lead to the formation of inflammatory complement mediators ( C3a and C5a ) and the membrane attack complex . Not shown is activation by collectins ...
... pathway , or by the alternative pathway itself ( see text ) . Both pathways converge and lead to the formation of inflammatory complement mediators ( C3a and C5a ) and the membrane attack complex . Not shown is activation by collectins ...
Page 89
... Pathway Phosphoinositide - 3 - Kinase Pathway Inositol - Lipid Pathway Cyclic Adenosine Monophosphate Pathway JAK / STAT Pathway Stress - Activated Signaling Pathway Transcription Factors CELL CYCLE AND THE REGULATION OF CELL DIVISION ...
... Pathway Phosphoinositide - 3 - Kinase Pathway Inositol - Lipid Pathway Cyclic Adenosine Monophosphate Pathway JAK / STAT Pathway Stress - Activated Signaling Pathway Transcription Factors CELL CYCLE AND THE REGULATION OF CELL DIVISION ...
Page 93
... pathway IP3 release Ca2 + release Transcription factor activation Simplified overview of the major types of cell surface receptors and the principal signal transduction pathways ( see text ) . Signaling events from the tyrosine kinase ...
... pathway IP3 release Ca2 + release Transcription factor activation Simplified overview of the major types of cell surface receptors and the principal signal transduction pathways ( see text ) . Signaling events from the tyrosine kinase ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
18 other sections not shown
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin