Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
Results 1-3 of 74
Page 16
... necrosis . A , Kidney infarct exhibiting. from the progressive degradative action of enzymes on the lethally injured cell ( cells placed immediately in fixative are dead but not necrotic ) . As commonly used , necrosis is the gross and ...
... necrosis . A , Kidney infarct exhibiting. from the progressive degradative action of enzymes on the lethally injured cell ( cells placed immediately in fixative are dead but not necrotic ) . As commonly used , necrosis is the gross and ...
Page 17
... necrosis . A , Kidney infarct exhibiting coagulative necrosis , with loss of nuclei and clumping of cytoplasm but with preservation of basic outlines of glomerular and tubular architecture . B , A focus of liquefactive necrosis in the ...
... necrosis . A , Kidney infarct exhibiting coagulative necrosis , with loss of nuclei and clumping of cytoplasm but with preservation of basic outlines of glomerular and tubular architecture . B , A focus of liquefactive necrosis in the ...
Page 848
... necrosis may be limited to scattered cells within hepatic lobules ( focal necrosis ) or to the interface be- tween the periportal parenchyma and inflamed portal tracts ( interface hepatitis ) . With more se- vere inflammatory injury ...
... necrosis may be limited to scattered cells within hepatic lobules ( focal necrosis ) or to the interface be- tween the periportal parenchyma and inflamed portal tracts ( interface hepatitis ) . With more se- vere inflammatory injury ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
18 other sections not shown
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin