Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 153
... lysosomal enzymes ( or acid hydrolases , as they are sometimes called ) are syn- thesized in the endoplasmic reticulum and transported to the Golgi apparatus . Within the Golgi complex , they un- dergo a variety of post - translational ...
... lysosomal enzymes ( or acid hydrolases , as they are sometimes called ) are syn- thesized in the endoplasmic reticulum and transported to the Golgi apparatus . Within the Golgi complex , they un- dergo a variety of post - translational ...
Page 154
... ( lysosomal glucosidase ) GM. Complex substrate Normal lysosomal degradation B Small diffusible end products Lysosomal enzyme deficiency B Figure 6-12 Schematic diagram illustrating the pathogenesis of lysosomal storage dis- eases . In the ...
... ( lysosomal glucosidase ) GM. Complex substrate Normal lysosomal degradation B Small diffusible end products Lysosomal enzyme deficiency B Figure 6-12 Schematic diagram illustrating the pathogenesis of lysosomal storage dis- eases . In the ...
Page 155
... lysosomes but are secreted extracellularly Aspartylglycosamine amide hydrolase Acid lipase Lysosomal acid phosphatase N - acetylgalactosamine - galactose - glucose- ceramide Cleavage site. Table 6-5 . LYSOSOMAL STORAGE DISEASES Disease ...
... lysosomes but are secreted extracellularly Aspartylglycosamine amide hydrolase Acid lipase Lysosomal acid phosphatase N - acetylgalactosamine - galactose - glucose- ceramide Cleavage site. Table 6-5 . LYSOSOMAL STORAGE DISEASES Disease ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin