Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Results 1-3 of 70
Page 484
... lesions indistinguishable from fibrosarco- mas occurring in adults . Biologic behavior cannot be predicted based on histology alone , however , because some of the lesions ( including the cellular fibromatoses or infantile fibrosarcomas ) ...
... lesions indistinguishable from fibrosarco- mas occurring in adults . Biologic behavior cannot be predicted based on histology alone , however , because some of the lesions ( including the cellular fibromatoses or infantile fibrosarcomas ) ...
Page 759
... lesions that rarely become bullous and ulcerated ; seen in more than 50 % of patients with cutaneous lichen planus and , rarely , is the sole manifestation Usually vulgaris ; vesicles and bullae prone to rupture , leaving hyperemic ...
... lesions that rarely become bullous and ulcerated ; seen in more than 50 % of patients with cutaneous lichen planus and , rarely , is the sole manifestation Usually vulgaris ; vesicles and bullae prone to rupture , leaving hyperemic ...
Page 966
... lesions or with the nephrotic syndrome have a somewhat poorer prognosis , and renal failure occurs in those with the crescentic lesions . BACTERIAL ENDOCARDITIS Glomerular lesions occurring in the course of bacterial endocarditis ...
... lesions or with the nephrotic syndrome have a somewhat poorer prognosis , and renal failure occurs in those with the crescentic lesions . BACTERIAL ENDOCARDITIS Glomerular lesions occurring in the course of bacterial endocarditis ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin