Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 543
... Heart dis- ease is the predominant cause of disability and death in Left ventricular hypertrophy . A , Pressure hypertrophy due to. 13 NORMAL HEART MYOCARDIUM BLOOD SUPPLY VALVES EFFECTS OF AGING ON THE HEART PRINCIPLES OF CARDIAC ...
... Heart dis- ease is the predominant cause of disability and death in Left ventricular hypertrophy . A , Pressure hypertrophy due to. 13 NORMAL HEART MYOCARDIUM BLOOD SUPPLY VALVES EFFECTS OF AGING ON THE HEART PRINCIPLES OF CARDIAC ...
Page 544
... heart disease ( IHD ) , the most prevalent subgroup , is estimated to be in excess of $ 100 billion . The major categories of cardiac diseases considered in this chapter include obstructive coronary ar- tery disease ( also called IHD ) ...
... heart disease ( IHD ) , the most prevalent subgroup , is estimated to be in excess of $ 100 billion . The major categories of cardiac diseases considered in this chapter include obstructive coronary ar- tery disease ( also called IHD ) ...
Page 565
... heart disproportionately to the increase in overall cardiac size ( Fig . 13-12 ) . The left ventricular wall thickness may exceed 2.0 cm , and the heart weight may exceed 500 g . In time , the increased thickness of the left ventricular ...
... heart disproportionately to the increase in overall cardiac size ( Fig . 13-12 ) . The left ventricular wall thickness may exceed 2.0 cm , and the heart weight may exceed 500 g . In time , the increased thickness of the left ventricular ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
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abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin