Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Results 1-3 of 58
Page 83
... granulomas and epithelioid types Gumma : microscopic to grossly visible lesion , enclosing wall of histiocytes ; plasma cell infiltrate ; center cells are necrotic without loss of cellular outline Rounded or stellate granuloma ...
... granulomas and epithelioid types Gumma : microscopic to grossly visible lesion , enclosing wall of histiocytes ; plasma cell infiltrate ; center cells are necrotic without loss of cellular outline Rounded or stellate granuloma ...
Page 351
... Granulomas of secondary tuberculosis most often occur in the apex of the lungs but may be widely disseminated in the lungs , kidneys , meninges , marrow , and other organs . These granulomas , which fail to contain the spread of the ...
... Granulomas of secondary tuberculosis most often occur in the apex of the lungs but may be widely disseminated in the lungs , kidneys , meninges , marrow , and other organs . These granulomas , which fail to contain the spread of the ...
Page 522
... granulomas , and infiltration of vessels and perivascular tissues by eosinophils is striking . Severe renal disease is infrequent . Coronary arteritis and myocar- ditis are the principal causes of morbidity and mortality . p - ANCA are ...
... granulomas , and infiltration of vessels and perivascular tissues by eosinophils is striking . Severe renal disease is infrequent . Coronary arteritis and myocar- ditis are the principal causes of morbidity and mortality . p - ANCA are ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin