Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 140
... gene , to clone it , and ultimately to determine the molecular changes that affect the gene in patients with the disorder . Because in many common single - gene disorders , such as cystic fibrosis , there was no clue to the nature of ...
... gene , to clone it , and ultimately to determine the molecular changes that affect the gene in patients with the disorder . Because in many common single - gene disorders , such as cystic fibrosis , there was no clue to the nature of ...
Page 285
... gene has been mapped , as well as one of the three immunoglobulin gene - carrying chromosomes . At its normal locus , the expression of the myc gene is tightly controlled and is expressed only during certain stages of the cell cycle ...
... gene has been mapped , as well as one of the three immunoglobulin gene - carrying chromosomes . At its normal locus , the expression of the myc gene is tightly controlled and is expressed only during certain stages of the cell cycle ...
Page 292
... gene . If the p53 gene is mutated , however , the hypoxic tumor cells are resistant to apoptosis . Thus , hypoxia selects for cells in which the p53 gene is inactive , and propagation of p53- deficient cells is favored . In addition to ...
... gene . If the p53 gene is mutated , however , the hypoxic tumor cells are resistant to apoptosis . Thus , hypoxia selects for cells in which the p53 gene is inactive , and propagation of p53- deficient cells is favored . In addition to ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin