Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Results 1-3 of 76
Page 106
... fibroblast ( and endothelial cell ) ingrowth . Migration of fibroblasts to the site of injury and their subsequent proliferation are triggered by multiple growth factors , including TGF - ß , PDGF , EGF , FGF , and the so- called ...
... fibroblast ( and endothelial cell ) ingrowth . Migration of fibroblasts to the site of injury and their subsequent proliferation are triggered by multiple growth factors , including TGF - ß , PDGF , EGF , FGF , and the so- called ...
Page 227
... fibroblasts or collagen . There seems to be a general con- sensus among experts that fibrosis is secondary to abnor- mal activation of the immune system.70 It is proposed that CD4 + T cells responding to an as yet unidentified antigen ...
... fibroblasts or collagen . There seems to be a general con- sensus among experts that fibrosis is secondary to abnor- mal activation of the immune system.70 It is proposed that CD4 + T cells responding to an as yet unidentified antigen ...
Page 1188
... fibroblasts within the dermis . Note the characteristic overlying epidermal hyperplasia and the tendency of fibroblasts to surround individual collagen bundles . MORPHOLOGY . The most common form of fibrous histiocytoma is referred to ...
... fibroblasts within the dermis . Note the characteristic overlying epidermal hyperplasia and the tendency of fibroblasts to surround individual collagen bundles . MORPHOLOGY . The most common form of fibrous histiocytoma is referred to ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin