Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Results 1-3 of 72
Page 249
... develop non - Hodgkin lymphoma has increased stead- ily . It is currently believed that approximately 6 % of all patients with AIDS develop lymphoma during their life- time . Thus , the risk of developing non - Hodgkin lymphoma is ...
... develop non - Hodgkin lymphoma has increased stead- ily . It is currently believed that approximately 6 % of all patients with AIDS develop lymphoma during their life- time . Thus , the risk of developing non - Hodgkin lymphoma is ...
Page 318
... develop an EBV infection , it does not take the usual self - limited form of infectious mononucleo- sis but in the vast majority of cases evolves into a fatal form of infectious mononucleosis . Approximately 25 % of XLP patients develop ...
... develop an EBV infection , it does not take the usual self - limited form of infectious mononucleo- sis but in the vast majority of cases evolves into a fatal form of infectious mononucleosis . Approximately 25 % of XLP patients develop ...
Page 1279
... develop decreased sensation in the distal extremities with comparably less evident motor abnormali- ties . The loss of pain sensation can result in the develop- ment of ulcers that heal poorly because of the diffuse vascular injury in ...
... develop decreased sensation in the distal extremities with comparably less evident motor abnormali- ties . The loss of pain sensation can result in the develop- ment of ulcers that heal poorly because of the diffuse vascular injury in ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
18 other sections not shown
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin