Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Page 526
... aortic wall.72 , 73 There is also evidence that matrix metalloproteinases ( MMPs ) and plasminogen activators , which degrade extra- cellular matrix ( ECM ) ( Chapter 4 ) , contribute to aneurysm formation.74 Clinical Course . The ...
... aortic wall.72 , 73 There is also evidence that matrix metalloproteinases ( MMPs ) and plasminogen activators , which degrade extra- cellular matrix ( ECM ) ( Chapter 4 ) , contribute to aneurysm formation.74 Clinical Course . The ...
Page 567
... aortic stenosis of a previously normal valve having three cusps ( viewed from the aortic aspect ) . Nodular masses of calcium are heaped up ... aortic stenosis . Aortic stenosis comes to clinical attention primarily. Chapter 13 567 THE HEART.
... aortic stenosis of a previously normal valve having three cusps ( viewed from the aortic aspect ) . Nodular masses of calcium are heaped up ... aortic stenosis . Aortic stenosis comes to clinical attention primarily. Chapter 13 567 THE HEART.
Page 568
... aortic stenosis to describe the latter condition . MORPHOLOGY . The morphologic hallmark of non- rheumatic , calcific aortic stenosis ( with either tri- cuspid or bicuspid valves ) is heaped - up calcified masses within the aortic cusps ...
... aortic stenosis to describe the latter condition . MORPHOLOGY . The morphologic hallmark of non- rheumatic , calcific aortic stenosis ( with either tri- cuspid or bicuspid valves ) is heaped - up calcified masses within the aortic cusps ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin