Robbins Pathologic Basis of DiseasePresenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
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Results 1-3 of 95
Page 62
... ACTIVATION In addition to stimulating locomotion , many chemotactic factors , particularly when in high concentrations , induce other responses in the leukocytes , referred to under the rubric of leukocyte activation ( see Fig . 3-11 ) ...
... ACTIVATION In addition to stimulating locomotion , many chemotactic factors , particularly when in high concentrations , induce other responses in the leukocytes , referred to under the rubric of leukocyte activation ( see Fig . 3-11 ) ...
Page 281
... Activation of transcription myc protein Model for action of ras genes . When a normal cell is stimulated through a growth factor receptor , inactive ( GDP - bound ) ras is activated to a GTP - bound state . Activated ras recruits raf ...
... Activation of transcription myc protein Model for action of ras genes . When a normal cell is stimulated through a growth factor receptor , inactive ( GDP - bound ) ras is activated to a GTP - bound state . Activated ras recruits raf ...
Page 640
... activation of the coagulation sequence that leads to the formation of mi- crothrombi throughout the microcirculation of the body , but often in a quixotically uneven distribution . Sometimes the coagulopathy is localized to a specific ...
... activation of the coagulation sequence that leads to the formation of mi- crothrombi throughout the microcirculation of the body , but often in a quixotically uneven distribution . Sometimes the coagulopathy is localized to a specific ...
Contents
GENERAL PATHOLOGY | 1 |
6 | 26 |
Acute and Chronic Inflammation | 50 |
Copyright | |
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Common terms and phrases
abnormal acid activation acute adhesion amyloidosis angiogenesis antibodies antigens apoptosis associated bacteria bind blood cancer carcinogens carcinoma cause CD4+ cell cycle cell death cellular Chapter chemical chromosome chronic clinical collagen colon common cyclin cytokines damage deficiency develop disease disorders drugs edema effects endothelial cells endothelium enzymes epithelial cells extracellular matrix fever fibrosis Figure function gene genetic granulomas growth factor hepatitis host cells human immune increased induce infection infiltrate inflammation inflammatory integrins intracellular involved kinase lesions leukocytes liver lung lymph nodes lymphocytes lymphoma lysosomal macrophages malignant matrix mechanisms mediators membrane metabolism molecular molecules MORPHOLOGY muscle mutations necrosis neoplasms nervous system neutrophils normal occur oncogenes organisms oxidative parasites pathogenesis pathogens pathway patients peptide phagocytosis plasma platelet produce proliferation protein reactions receptor release renal respiratory response result signals skin surface syndrome synthesis T-cell tion tissue toxic toxin tumor tumor cells vascular viral virus viruses vitamin