Bioimaging in Neurodegeneration
Patricia A. Broderick, David N. Rahni, Edwin H. Kolodny
Springer Science & Business Media, Nov 5, 2007 - Medical - 314 pages
Bioimaging is in the forefront of medicine for the diagnosis and helps to predict the progression of AD via mild cognitive treatment of neurodegenerative disease. Conventional magnetic impairment (MCI) studies. resonance imaging (MRI) uses interactive external magnetic fields Novel neuroimaging technologies, such as neuromolecular and resonant frequencies of protons from water molecules. imaging (NMI) with a series of newly developed BRODERICK « However, newer sequences, such as magnetization-prepared rapid PROBE sensors, directly image neurotransmitters, precursors, acquisition gradient echo (MPRAGE), are able to seek higher and metabolites in vivo, in real time and within seconds, at separate levels of anatomic resolution by allowing more rapid temporal and selective waveform potentials. NMI, which uses an imaging. Magnetic resonance spectroscopy (MRS) images electrochemical basis for detection, enables the differentiation of metabolic changes, enabling underlying pathophysiologic neurodegenerative diseases in patients who present with mesial dysfunction in neurodegeneration to be deciphered. Neuro- versus neocortical temporal lobe epilepsy. In fact, NMI has some 1 chemicals visible with proton H MRS include N-acetyl aspartate remarkable similarities to MRI insofar as there is technological (NAA), creatine/phosphocreatine (Cr), and choline (Cho); NAA dependence on electron and proton transfer, respectively, and is considered to act as an in vivo marker for neuronal loss and/or further dependence is seen in both NMI and MRI on tissue neuronal dysfunction. By extending imaging to the study of composition such as lipids.
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abnormalities acid activity adrenoleukodystrophy Alzheimer Alzheimer’s disease amyloid Ann Neurol areas atrophy basal ganglia binding biochemical blood flow brain Broderick cell cerebral changes choline clinical cognitive correlation cortex cortical dysplasia decreased deficiency dementia demonstrated demyelination detection diagnosis diffuse disorders dopamine transporter dopaminergic enzyme epileptic epileptogenic evaluation FDG PET FDOPA uptake fibrillization focal frontal function gene genetic glucose gray matter hippocampal human ictal increased interictal intraoperative involvement L-Trp lactate lesions leukodystrophy magnetic resonance imaging magnetic resonance spectroscopy metabolism metabolites mitochondrial motor multiple system atrophy mutations myelin neocortex neocortical Neurodegeneration neuroimaging Neurology neurons Neurosci neurotoxicity normal Nucl onset optical oxidation Parkinson’s disease pathology PD patients peptide PET study plaques positron emission tomography progression protein proton putamen receptor regions reported resection scan seizures serotonin signal SPECT striatal striatum surgery syndrome T2-weighted temporal lobe epilepsy tion tissue tracer transplantation visual vitro vivo white matter