Robbins' Pathologic Basis of Disease, Volume 1Presenting the latest edition of this popular, comprehensive and practical text of pathology. Written with great clarity for easy readability, this reference offers detailed discussions of genetic disorders, cellular injury and death, neoplasia, the skeletal system and soft tissue tumors, and much more. Completely revised and updated, this edition is even more user-friendly with the use of text boxes for key topics in each chapter and a new full-color design. Features 1200 excellent 4-color illustrations! |
From inside the book
Results 1-3 of 88
Page 158
... NORMAL PROBE Normal. 5 6 Amino acids Codons Pro Val CCT 7 Glu HbS ↑ Mst II recognition sequence сст Pro GAG Glu HbA Sickle mutation . ẞs globin Mst II Mst II 1.35 Kb cDNA probe BA globin Mst II 1.15 Kb Mst II Mst II 0.2 Kb Southern blot ...
... NORMAL PROBE Normal. 5 6 Amino acids Codons Pro Val CCT 7 Glu HbS ↑ Mst II recognition sequence сст Pro GAG Glu HbA Sickle mutation . ẞs globin Mst II Mst II 1.35 Kb cDNA probe BA globin Mst II 1.15 Kb Mst II Mst II 0.2 Kb Southern blot ...
Page 159
... NORMAL PROBE Normal Heterozygote Homozygote MZ ZZ MM SOUTHERN BLOT ANALYSIS Normal gene BB BB AA AB Affected Affected Normal Carrier. carry somes that the CF gene . It follows , therefore , that the success of such a strategy depends ...
... NORMAL PROBE Normal Heterozygote Homozygote MZ ZZ MM SOUTHERN BLOT ANALYSIS Normal gene BB BB AA AB Affected Affected Normal Carrier. carry somes that the CF gene . It follows , therefore , that the success of such a strategy depends ...
Page 291
... normal alleles of the Rb locus must be inacti- vated ( " two hits " ) for the development of retinoblas- toma ( Fig . 6-34 ) . In familial cases , children are born with one normal and one defective copy of the Rb gene . They lose the ...
... normal alleles of the Rb locus must be inacti- vated ( " two hits " ) for the development of retinoblas- toma ( Fig . 6-34 ) . In familial cases , children are born with one normal and one defective copy of the Rb gene . They lose the ...
Contents
Cellular Injury and Adaptation | 1 |
Inflammation and Repair | 39 |
Fluid and Hemodynamic Derangements | 87 |
Copyright | |
28 other sections not shown
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abnormal acid activity acute adult agents amyloid amyloidosis antibodies antigens arteries atherosclerosis bacterial blood bone cancer carcinogens carcinoma cardiac cause cellular changes chemical chromosome chronic clinical collagen complex damage death defects deficiency develop diagnosis disease disorders drugs edema effects endothelium Engl enzymes epithelial fatty fever fibroblasts fibrosis Figure fluid function gene genetic granulomas growth factor hemorrhage hepatic histologic host human immune immunologic increased induce infants infarction infection infiltration inflammation inflammatory injury interstitial intestinal intracellular involved lesions leukemia leukocytes levels lipid liver lung lymph nodes lymphocytes lysosomal macrophages malignant mechanisms mediators membrane metabolism monocytes mucosa muscle mutations necrosis necrotic neoplasms neutrophils normal occur oncogenes organs parasites pathogenesis patients phagocytic phagocytosis plasma platelet produce proliferation protein pulmonary reaction receptor renal respiratory response result risk serum skin specific syndrome synthesis tion tissue toxic tumor tumor cells ulcer usually vascular vessels viral virus viruses vitamin vitamin D